Deciphering plant-pathogen communication: fresh perspectives for molecular resistance breeding.

By K. Hammond-Kosack, J. Parker. Current Opinion in Biotechnology, 2003

650 citations recorded by the source corpus.

Subfields: Molecular Biology, Plant Science

View Paper: https://www.semanticscholar.org/paper/ffb4b61c47f3bc4d0e8ff1639071903563d86b94

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Citations in the corpus

Marker assisted selection in crop plants (2005, neutral): “R genes against a variety of different pathogens have now been cloned from a variety of crop plants (Baker et al., 1997; Hammond-Kosack and Parker, 2003).”
Elicitor recognition, signal transduction and induced resistance in plants (2012, background): “Many pathogen components can be recognized by plants, including Avr genes, structural components from bacteria such as flagellin, toxins, Hrp proteins delivered by the bacterial type III secretion system, cell-wall components such as chitin and melanin, enzymes that degrade plant polymers such as pe”
Plant immunity: the EDS1 regulatory node. (2005, background): “A large catalogue of Arabidopsis mutants that are compromised in basal defenses to virulent pathogens points to the involvement of many genes in maintaining this resistance layer and to the existence of numerous potential targets that the pathogen might disable to promote disease [2].”
Reactive Oxygen Species in Plant–Pathogen Interactions (2009, background): “characterised in several different plant species against important microbial pathogens (Hammond-Kosack and Parker 2003 ; van’t Slot and Knogge 2002) .”
Poplar and Pathogen Interactions: Insights from Populus Genome-Wide Analyses of Resistance and Defense Gene Families and Gene Expression Profiling (2009, background): “These include a genetically determined pathogen recognition system controlled by host resistance (R) genes (Hammond-Kosack and Parker, 2003; Jones and Dangl, 2006).”
Advances in plant disease and pest management (2010, background): “Only through field and non-field trialling can researchers and commercial plant breeders test the potential effectiveness of each R gene in a specific plant genetic background (Hammond-Kosack & Parker 2003).”
Plant NBS-LRR proteins: adaptable guards (2006, background): “Plant NBS-LRR proteins act through a network of signaling pathways and induce a series of plant defense responses, such as activation of an oxidative burst, calcium and ion fluxes, mitogen-associated protein kinase cascade, induction of pathogenesis-related genes, and the hypersensitive response [4,”
Silverleaf Whitefly Induces Salicylic Acid Defenses and Suppresses Effectual Jasmonic Acid Defenses1[W][OA] (2006, background): “While some pathogens evade host defenses by actively catabolizing antimicrobial compounds (Bouarab et al., 2002), there is a growing evidence that plant pathogens produce effectors that antagonize defense signaling networks (Hammond-Kosack and Parker, 2003; Kamoun, 2006).”
Redox Regulation in Photosynthetic Organisms: Signaling, Acclimation, and Practical Implications (2009, positive): “…facilitates the pre-acclimation of photosynthetic tissues to high light is distinct from that of pathogen-related systemic acquired resistance (359), these studies show that PAD4, EDS1, and MPK4 interact with LSD1 in signal pathways, leading to PCD (434) and with some responses to EEE (259, 260).”
Elicitors, effectors, and R genes: the new paradigm and a lifetime supply of questions. (2007, background): “This review does not cover our growing knowledge of genes that function “downstream” of R genes, but many are now known (31, 55, 70, 88, 185).”
Antibiotic resistance plasmids in wastewater treatment plants and their possible dissemination into the environment (2010, background): “Significant progress has been made in understanding the molecular mechanisms governing resistance to plant diseases and therefore could reduce pesticide use, which would conserve energy and provide farmers with new opportunities for sustainable disease control (Hammond-Kosack and Parker, 2003).”
Can Robigus defeat an old enemy? – Yellow rust of wheat (2005, background): “There is now considerable evidence that avirulence genes are pathogenicity factors, required by the pathogen to infect and establish a compatible association with its host (Hammond-Kosack & Parker 2003).”