Plant immunity: the EDS1 regulatory node.

By M. Wiermer, B. Feys, J. Parker. Current opinion in plant biology, 2005

628 citations recorded by the source corpus.

Subfields: Plant Science, Public Health, Environmental and Occupational Health

View Paper: https://www.semanticscholar.org/paper/0ee0289bc6ff19787780297eea5c4e302013dbfe

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Citations in the corpus

Emerging microbial biocontrol strategies for plant pathogens. (2018, neutral): “These pathogens have evolved specialized structures such as nutrient-absorbing haustoria that penetrate into the plasma membrane of the host cells and take up the nutrient sap and release e ﬀ ector molecules that let them grow invasively on particular host genotypes [85 – 88].”
Signaling in Plant-Microbe Interactions (2013, background): “For example, EDS1 (ENHANCED DISEASE SUSCEPTIBILITY 1) of Arabidopsis is required for TIR-NB-LRR mediated signaling (Wiermer et al. 2005) whereas NDR1 (NONRACE-SPECIFIC DISEASE RESISTANCE 1) is required for some of the CC-NB-LRR mediated signaling (Day et al. 2006).”
Plant immunity: towards an integrated view of plant–pathogen interactions (2010, background): “ENHANCED DISEASE SUSCEPTIBILITY 1 (EDS1) is required for signalling of all TIR-NB-LRRs tested to date, suggesting that it acts specifically in TIR domain signallin”
Hormonal modulation of plant immunity. (2012, background): “EDS1 and PAD4, which act early in the SA pathway and stimulate SA biosynthesis, were identified as downstream components of MPK4 function (Brodersen et al. 2006).”
The hypersensitive response; the centenary is upon us but how much do we know? (2008, background): “One node is dependent on EDS1 and PAD4 which is required for the function of the TIR-NBS-LRR class of R genes (reviewed by Wiermer et al., 2005).”
Potent Induction of Arabidopsis thaliana Flowering by Elevated Growth Temperature (2006, positive): “More recently, EDS1 has been shown to play important roles in responses to several abiotic stresses as well (reviewed in [49]).”
Salicylic Acid Biosynthesis and Metabolism (2011, methodology): “In addition to R protein-mediated signaling, NDR1, EDS1, PAD4 and SAG101 have been implicated in PTI induced by virulent pathogens or PAMP treatment (Century et al, 1995; Wiermer et al, 2005; Vlot et al, 2009; Knepper et al, 2011).”
Salicylic Acid, a multifaceted hormone to combat disease. (2009, background): “The lipase-like protein ENHANCED DISEASE SUSCEPTIBILITY 1 (EDS1) and its sequence-related interacting partner PHYTOALEXIN DEFICIENT 4 (PAD4) act upstream of SA in basal resistance to host-adapted biotrophic pathogens as well as in ETI initiated by the TIR-NBSLRR subset of R genes (1, 213).”
Magical mystery tour: Salicylic acid signalling (2015, background): “Indeed, the action of RPS4 (Resistance to Pseudomonas syringae 4), a typical TIR-NBS-LRR, requires EDS1 to induce SA-dependent and SA-independent resistance to pathogens (Feys et al., 2005; Garcia et al., 2010; Wiermer et al., 2005; Wirthmueller et al., 2007).”