Stabilization of atherosclerotic plaques: New mechanisms and clinical targets

By Peter Libby, M. Aikawa. Nature Medicine, 2002

613 citations recorded by the source corpus.

Subfields: Cancer Research, Immunology, Surgery

View Paper: https://www.semanticscholar.org/paper/fb5ee648f7810aa9d077a7acf6df45c8ce049bd9

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Cancer Research

1. A. Newby
2. G. Hansson
3. M. Levi
4. T. van der Poll
5. Peter Libby

In Immunology

1. Peter Libby
2. G. Hansson
3. A. Newby
4. T. van der Poll
5. M. Levi
6. P. Ridker
7. I. Tabas
8. V. Z. Rocha

In Surgery

1. G. Hansson
2. Peter Libby
3. A. Newby
4. T. van der Poll
5. M. Levi
6. I. Tabas
7. A. Lichtman

Citations in the corpus

• Bidirectional Relation Between Inflammation and Coagulation (2004, background): “Amplification loops consist of (1) the activation of factor IX by the tissue factor–factor VIIa complex, generating large amounts of additional factor Xa, (2) activation of the essential”
• The immune response in atherosclerosis: a double-edged sword (2006, background): “Adaptive immunity disrupts plaques In general, the gravest clinical complications of atherosclerosis result from the sudden thrombotic occlusion of an arter”
• Dual role of matrix metalloproteinases (matrixins) in intimal thickening and atherosclerotic plaque rupture. (2005, background): “cyclical strain caused by the cardiac cycle (158).”
• Inflammation and coagulation (2010, background): “Inflammatory cells in atherosclerotic plaques produce abundant tissue factor, and on plaque rupture there is extensive tissue factor exposure to blood (9).”