Bidirectional Relation Between Inflammation and Coagulation

By M. Levi, T. van der Poll, H. Büller. Circulation, 2004

927 citations recorded by the source corpus.

Subfields: Hematology, Internal Medicine, Pulmonary and Respiratory Medicine

View Paper: https://www.semanticscholar.org/paper/5af4ec93f207db98e2ea1f49c9db10b691144996

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In Hematology

1. M. Levi

Citations in the corpus

• Inflammation, endothelium, and coagulation in sepsis (2008, background): “modulate the inflammatory response [7, 8].”
• Platelets, inflammation and tissue regeneration (2011, background): “Studies using PAR-4 deficient mice showed a markedly lower inflammatory response to soluble TF despite the fact that binding of TF to PAR-2 can result in the up-regulation of inflammatory rersponses in macrophages and can affect neutrophil infiltration and TNF-α production ([132], reviewed in [109])”
• Molecular Intercommunication between the Complement and Coagulation Systems (2010, background): “Therefore, it is tempting to speculate whether various clotting factors might contribute locally and systemically to generate C3a and C5a, which, in turn act as chemoattractants for phagocytic cells to the site of inflammation, where these cells release their major arsenals of tissue-damaging protea”