Inflammation in prostate carcinogenesis

By A. M. Marzo, E. Platz, S. Sutcliffe, Jianfeng Xu, H. Grönberg, Charles G. Drake, Y. Nakai, W. Isaacs, W. Nelson. Nature Reviews. Cancer, 2007

1,697 citations recorded by the source corpus.

Subfields: Cancer Research, Molecular Biology, Oncology

View Paper: https://www.semanticscholar.org/paper/f5a96819a2c7612f7de4663079e4f9c12d2d7452

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Cancer Research

1. Carlo Maria Croce
2. George A. Calin
3. Chang-Gong Liu
4. Stefano Volinia
5. F. Petrocca
6. Marilena V. Iorio
7. Joshua T. Mendell
8. William C. S. Cho

In Molecular Biology

1. Carlo Maria Croce
2. George A. Calin
3. Chang-Gong Liu
4. Stefano Volinia
5. C. Abate-Shen
6. Prashanth Rawla
7. M. Shen
8. Martin J. Aryee

In Oncology

1. C. Abate-Shen
2. Carlo Maria Croce
3. Prashanth Rawla
4. George A. Calin
5. M. Shen

Citations in the corpus

• Genomic profiling of microRNA and mRNA reveals deregulated microRNA expression in prostate cancer (2008, background): “It is this function of miR-32 that could be the causal link between its altered expression and prostate cancer development because several of the known prostate cancer susceptibility genes are also involved in host defense (31).”
• Viral Encounters with 2′,5′-Oligoadenylate Synthetase and RNase L during the Interferon Antiviral Response (2007, background): “cancer (21), a role for XMRV in prostate cancer etiology, al-”
• Epidemiology of Prostate Cancer (2019, background): “Chronic inflammation causes proliferative inflammatory atrophy (PIA) [174], which may develop prostatic intraepithelial neoplasia (PIN) a well-known precursor of prostate cancer [247].”
• Molecular genetics of prostate cancer: new prospects for old challenges. (2010, background): “These observations have raised interest in the potential causes of prostate inflammation, which may include hormonal perturbations such as altered androgen and estrogen levels, or infection by bacterial or viral agents, physical trauma, or dietary factors (De Marzo et al. 2007b).”
• Androgen-induced TOP2B mediated double strand breaks and prostate cancer gene rearrangements (2010, background): “, or the inflammatory milieu often seen surrounding prostate preneoplastic lesions in vivo”
• Prostate cancer and inflammation: the evidence (2012, background): “environmental exposures.(2) The aim of this review is to summarize the current literature regarding a role for chronic inflammation in prostate cancer aetiology, with a specific focus on potential stimuli for prostatic inflammation, prostate cancer immunobiology, inflammatory pathways and cytokines ”