[Disseminated intravascular coagulation].
By T. Iba, A. Kidokoro. Nihon rinsho. Japanese journal of clinical medicine, 2003
1,502 citations recorded by the source corpus.
View Paper: https://www.semanticscholar.org/paper/f21421b98f2b06a43936f53ceeef82a68a231074
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Citations in the corpus
- Bidirectional Relation Between Inflammation and Coagulation (2004, background): “The assembly of several complexes, such as the “tenase complex,” consisting of factor X, factor IXa, factor VIIIa, and calcium, or the prothrombinase complex mentioned above will be markedly facilitated if a suitable phospholipid surface is available and occurs in vivo, presumably on the membrane of”
- New fundamentals in hemostasis. (2013, background): “The APC system plays a central role in the pathogenesis of disseminated intravascular coagulation (DIC) and associated organ dysfunction (85, 157, 158).”
- Non-canonical proteolytic activation of human prothrombin by subtilisin from Bacillus subtilis may shift the procoagulant–anticoagulant equilibrium toward thrombosis (2017, background): “strategies, the pathological activation of coagulation in infectious diseases is associated with high morbidity and mortality (22, 68).”
- Inflammation and coagulation (2010, background): “DIC has shown to be an independent predictor of organ failure and mortality (3, 24).”
- Plumb's Veterinary Drug Handbook (2005, unknown): “(Byars 1987) As adjunctive therapy in endotoxic shock: a) 40 Units/kg IV or SC 2 – 3 times a day may prevent the development of microthrombi; addi tional studies are required to confirm positive benefits (Semrad and Moore 1987) As adjunctive therapy in the prevention of laminitis: a) 25 – 100 Units/”
- Towards Definition, Clinical and Laboratory Criteria, and a Scoring System for Disseminated Intravascular Coagulation (2001, background): “Assessment of a low and/or decreasing platelet count more accurately reflects both consumption and thrombin generation and is helpful in establishing the presence and severity of DIC (10).”
- The coagulopathy of trauma: a review of mechanisms. (2008, background): “This phenomenon is not DIC as described in sepsis and other conditions.(85) It is marked by early onset, prolonged PT and PTT and a relative sparing of platelets and fibrinogen.”
- Guidelines for the diagnosis and management of disseminated intravascular coagulation (2009, unknown): “Measurement of fibrinogen has been widely advocated as a useful tool for the diagnosis of DIC but in fact is not very helpful in most cases (Levi & Ten, 1999).”