ATP-Dependent Chromatin-Remodeling Complexes
By M. Vignali, Ahmed H. Hassan, Kristen E. Neely, Jerry L. Workman. Molecular and Cellular Biology, 2000
793 citations recorded by the source corpus.
Subfields: Molecular Biology
View Paper: https://www.semanticscholar.org/paper/eb033ee8d813675e93f39602bdbc47622f247a5d
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Citations in the corpus
- Nuclear retinoid receptors and the transcription of retinoid-target genes. (2004, background): “…(SWI/SNF) which use the energy of ATP hydrolysis to reposition nucleosomes at the promoter through sliding them in cis or displacing them in trans, allowing the formation of nucleosome-free or nucleosome-spaced regions (Kingston and Narlikar, 1999; Vignali et al., 2000; Narlikar et al., 2002).”
- Nuclear hormone receptors and gene expression. (2001, background): “Two different mechanisms can alleviate this repression: histones can be posttranslationally modified to destabilize chromatin, and nucleosomes can be disrupted through activation of ATP-driven machines that can remodel chromatin (198, 276).”
- Chromatin remodelling: the industrial revolution of DNA around histones (2006, background): “SWI/SNF remodellers all contain a conserved ATPase subunit and a set of five additional, conserved core members that together define the famil”
- The SWI/SNF complex — chromatin and cancer (2004, background): “Existing biochemical data support a model by which interconversion of nucleosomes between basal and stably remodelled states leads to sliding of nucleosomes along the DNA helix and also to transfer of nucleosomes from one helix to anothe”
- Transcription regulation by histone methylation: interplay between different covalent modifications of the core histone tails. (2001, background): “One family includes multiprotein complexes that utilize the energy derived from ATP hydrolysis to mobilize or alter the structure of nucleosomes (Kingston and Narl-ikar 1999; Vignali et al. 2000).”