Neurotrophin secretion: current facts and future prospects.
By Volkmar Lessmann, K. Gottmann, M. Malcangio. Progress in neurobiology, 2003
678 citations recorded by the source corpus.
Subfields: Cellular and Molecular Neuroscience, Developmental Neuroscience, Molecular Biology
View Paper: https://www.semanticscholar.org/paper/e26a4903ee6431fa6c217eea170a7c212ee32981
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In Cellular and Molecular Neuroscience
In Developmental Neuroscience
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Citations in the corpus
- Transcranial pulsed ultrasound stimulates intact brain circuits. (2010, background): “Brain-derived neurotrophic factor (BDNF) is one of the most potent neuromodulators of hippocampal plasticity, and its expression/secretion is known to be regulated by neuronal activity (Lessmann et al., 2003; Poo, 2001).”
- Activation of p75NTR by proBDNF facilitates hippocampal long-term depression (2005, background): “Like other neurotrophins, BDNF is first synthesized as a precursor (proBDNF), which is proteolytically cleaved to form mature BDNF (mBDNF”
- Brain-Derived Neurotrophic Factor and Neuropsychiatric Disorders (2012, background): “ProBDNF signals through the low-affinity neurotrophin receptor p75 that is believed to be involved in apoptosis (Roux and Barker, 2002; Lessmann et al., 2003).”
- Brain-Derived Neurotrophic Factor: A Key Molecule for Memory in the Healthy and the Pathological Brain (2019, background): “Brain derived neurotrophic factor is synthesized as the precursor proBDNF, that can be stored in either dendrites or axons (Lessmann et al., 2003), and undergoes cleavage intra or extracellularly (Lee et al.”
- Cation-chloride cotransporters and neuronal function. (2009, background): “It is a well-known fact that the release of trophic factors is enhanced during synchronous neuronal activity (Lessmann et al., 2003), and one of the functions of the early spontaneous network events described above may be to promote the release of these substances.”
- TrkB signalling pathways in LTP and learning (2009, background): “Evidence for the activity-dependent regulation of BDNF transcription and secretio”
- Resting Microglia Directly Monitor the Functional State of Synapses In Vivo and Determine the Fate of Ischemic Terminals (2009, background): “Possible candidate attractant signals which could be released from synapses include glutamate, ATP, brain-derived growth factor, and chemokines (Lessmann et al., 2003; Haynes et al., 2006), in addition to a role of any electrical signals.”
- BDNF function in adult synaptic plasticity: the synaptic consolidation hypothesis. (2005, background): “secretory vesicles (Lessmann et al., 2003).”