Diabetes and mitochondrial function: role of hyperglycemia and oxidative stress.
By A. Rolo, C. Palmeira. Toxicology and Applied Pharmacology, 2006
1,040 citations recorded by the source corpus.
Subfields: Clinical Biochemistry, Molecular Biology, Physiology
View Paper: https://www.semanticscholar.org/paper/e1d0a6cf2123446babe8cb6dd12fc3e41c871a4a
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In Clinical Biochemistry
In Molecular Biology
In Physiology
Citations in the corpus
- Pathogenesis of Insulin Resistance in Skeletal Muscle (2010, result): “[114] A.”
- Diabetes associated cell stress and dysfunction: role of mitochondrial and non‐mitochondrial ROS production and activity (2007, background): “Although our understanding of how hyperglycaemia-induced oxidative stress ultimately leads to tissue damage has advanced considerably in recent years (Vincent et al. 2005; Rolo & Palmeira, 2006), effective therapeutic strategies to prevent or delay the development of this damage remain limited.”
- Role of Catalase in Oxidative Stress- and Age-Associated Degenerative Diseases (2019, background): “Ankita Nandi,1 Liang-Jun Yan ,2 Chandan Kumar Jana,3 and Nilanjana Das 1 1Department of Biotechnology, Visva-Bharati University, Santiniketan, West Bengal 731235, India 2Department of Pharmaceutical Sciences, UNT System College of Pharmacy, University of North Texas Health Science Center, Fort Worth”
- Mitochondrial Glutathione, a Key Survival Antioxidant (2009, background): “This depletion in reducing equivalents results in augmented sensitivity to oxidative stress (122).”