EMT: 2016.

By M. Nieto, R. Huang, R. Jackson, J. P. Thiery. Cell, 2016

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Epithelial-mesenchymal plasticity in cancer progression and metastasis (2019, background): “EMT plays essential functions during embryonic development by facilitating gastrulation, neural crest delamination as well as the generation of diverse cell and tissue types (Nieto et al., 2016).”
Oxidative Stress in Cancer (2020, background): “Thus, both CAFs and TAMs “cooperate” with neoplastic cells by contributing to a developmental program called EMT by helping to remodel the extracellular matrix (ECM), thereby stimulating tumor cell proliferation, tumor angiogenesis, immunosuppression and tumor invasion (Nieto et al., 2016).”
Integrin Signaling in Cancer: Mechanotransduction, Stemness, Epithelial Plasticity, and Therapeutic Resistance (2019, background): “Emerging evidence suggests that these intermediary stages are more reversible and more conducive to metastatic colonization compared with a full mesenchymal state (Lambert et al., 2017; Nieto et al., 2016).”