Nomenclature of voltage-gated calcium channels.

By E. A. Ertel, K. Campbell, M. Harpold, F. Hofmann, Y. Mori, E. Perez-Reyes, A. Schwartz, Terrance P. Snutch, T. Tanabe, L. Birnbaumer and 2 more. Neuron, 2000

904 citations recorded by the source corpus.

Subfields: Cellular and Molecular Neuroscience, Electrochemistry, Molecular Biology

View Paper: https://www.semanticscholar.org/paper/d735f3dac2e78de73e53cd12ce6acd00a42bd501

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Citations in the corpus

PATHOPHYSIOLOGY OF MIGRAINE (2006, neutral): “1 (P/Q) type voltage-gated calcium channel (Ertel et al, 2000) CACNA1A gene.”
The Calcium Channel α2δ-2 Subunit Partitions with CaV2.1 into Lipid Rafts in Cerebellum: Implications for Localization and Function (2006, background): “Mammalian genes encoding 10 1, four , and four 2 subunits have been identified (Catterall, 2000; Ertel et al., 2000; Burgess et al., 2001).”
Resurgence of sodium channel research. (2001, methodology): “This nomenclature is similar to the ones that were developed for potassium (5) and calcium (6) channels, and it has been used for the purposes of this review.”
Voltage-gated calcium channels. (2011, background): “In cardiac, smooth, and skeletal muscle, the major Ca2þ currents are distinguished by high voltage of activation, large single channel conductance, slow voltage-dependent inactivation, marked up-regulation by cAMP-dependent protein phosphorylation pathways, and specific inhibition by Ca2þ…”
Cav1.3 (α1D) Ca2+ Currents in Neonatal Outer Hair Cells of Mice (2003, background): “3_/_, for nomenclature see Ertel et al. 2000).”
International Union of Pharmacology. XLVII. Nomenclature and Structure-Function Relationships of Voltage-Gated Sodium Channels (2005, background): “For potassium channels and calcium channels, all members of distinct subfamilies are less than 50% identical to those of other families, and there is much closer sequence similarity within families (Chandy and Gutman, 1993; Ertel et al., 2000).”
Voltage-gated ion channels and gating modifier toxins. (2007, background): “The Ca 1Ertel et al., 2000 V subfamily (Ca 1.1 to Ca 1.4) conduct L-type calcium currents that initiate contraction, endocrine secretion, and synaptic transmission atV V the specialized ribbon synapses involved in sensory input in the eye and ear ( ; ).”
Ca(V)1.2 calcium channel dysfunction causes a multisystem disorder including arrhythmia and autism. (2004, background): “Relief of inactivation was greater for G406Rpressed in heart, brain, smooth muscle, and pituitary and adrenal glands (Ertel et al., 2000).”