Voltage-gated calcium channels.
By William A. Catterall. Cold Spring Harbor Perspectives in Biology, 2011
1,596 citations recorded by the source corpus.
Subfields: Cellular and Molecular Neuroscience, Molecular Biology
View Paper: https://www.semanticscholar.org/paper/f6ad148b5b93b7497ae3d3d01e1f7a829cd86ed9
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In Cellular and Molecular Neuroscience
In Molecular Biology
Citations in the corpus
- Ion Channels in Innate and Adaptive Immunity (2015, background): “They are grouped into three subfamilies (CaV1, CaV2, CaV3) that contain 10 pore-forming α subunits and many associated regulatory β, γ, and δ subunits (67).”
- Macrophages Facilitate Electrical Conduction in the Heart (2017, neutral): “While depolarization of the resting membrane potential in working cardiomyocytes can impair excitation and conduction due to sodium channel inactivation, depolarization of AV nodal cells depends chiefly on calcium channels, which have less prominent voltage-dependent inactivation (Catterall, 2011).”
- Neuronal calcium signaling: function and dysfunction (2014, background): “The Cav3 subfamily is responsible for the T-type current and is important for the pacemaking and repetitive firing of action potentials in cardiac myocytes and thalamic neurons [10].”