International Union of Pharmacology. XLVII. Nomenclature and Structure-Function Relationships of Voltage-Gated Sodium Channels

By William A. Catterall, A. L. Goldin, Stephen G. Waxman. Pharmacological Reviews, 2005

1,284 citations recorded by the source corpus.

Subfields: Cellular and Molecular Neuroscience, Molecular Biology

View Paper: https://www.semanticscholar.org/paper/25270b3ed8cdbad0452469f89b87fff0bbf4e484

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In Cellular and Molecular Neuroscience

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Citations in the corpus

• FOXG1-dependent dysregulation of GABA/glutamate neuron differentiation in autism spectrum disorders (2015, positive): “8 mV, a phenotype most consistent with the Na v 1.1 brain sodium channel isoform (Catterall et al., 2005). size of sodium currents in control-and proband-derived neurons was similar (Figure S3), there was substantial cellto-cell variation in the voltage dependence of activation and inactivation,…”
• Enterochromaffin Cells Are Gut Chemosensors that Couple to Sensory Neural Pathways. (2017, positive): “Moreover, depolarization-evoked Ca2+ re- 186 Cell 170, 185–198, June 29, 2017 sponseswere inhibited byu-agatoxin IVA, which blocks presynaptic P/Q-type CaV channels (Catterall et al., 2005b) (Figures 1H and 1I).”