Adipose tissue dysfunction in obesity.

By M. Blüher. Experimental and clinical endocrinology & diabetes, 2009

602 citations recorded by the source corpus.

Subfields: Cardiology and Cardiovascular Medicine, Epidemiology, Physiology

View Paper: https://www.semanticscholar.org/paper/d358af76115434494b32830ac51d0e91aff2577b

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Cardiology and Cardiovascular Medicine

1. P. Trayhurn
2. F. Alsaraj
3. E. Rosen
4. M. Blüher

In Epidemiology

1. P. Trayhurn
2. F. Alsaraj
3. E. Rosen
4. M. Blüher
5. G. Hotamışlıgil
6. B. Spiegelman
7. Haiming Cao

In Physiology

1. P. Trayhurn
2. F. Alsaraj
3. E. Rosen
4. M. Blüher
5. G. Hotamışlıgil
6. B. Spiegelman
7. Haiming Cao

Citations in the corpus

• Metabolically Healthy Obesity (2020, methodology): “Signals from adipose tissue may include peptide hormones (adipokines), immune cells, and metabolites, which either specifically or as a pattern contribute to the development of type 2 diabetes, fatty liver disease, endothelial dysfunction, and cardiovascular diseases (57, 60, 64).”
• Hypoxia and adipose tissue function and dysfunction in obesity. (2013, background): “Another key development is the realization that adipose tissue depots become “inflamed” in obesity, with the view that the inflammatory state leads to the initiation and progression of the major obesity-associated diseases (3, 9, 81, 167, 228).”
• Transcriptional control of adipose lipid handling by IRF4 (2011, background): “The current epidemic of obesity has prompted intense investigation into the molecular mechanisms underlying fat storage and utilization (Bluher, 2009; Jensen, 2008).”
• Insulin-sensitive obesity. (2010, background): “Both genetic and environmental factors could contribute to increased ectopic visceral fat accumulation and hypertrophy of adipocytes (3), and this condition might represent the inability of an obese patient to become more (subcutaneously) obese (40).”
• Metabolic Syndrome and Insulin Resistance: Underlying Causes and Modification by Exercise Training (2013, unknown): “Much of this work is reviewed in references (71, 229, 515, 521, 669, 732).”
• Pathogenesis of Type 2 Diabetes Mellitus (2015, background): “Obesity, IR, and T2DM are growing health concerns, and the incidence and the prevalence of these diseases are increasing worldwide [32, 33].”