Formyl-peptide receptors revisited.

By Y. Le, P. Murphy, Ji Ming Wang. Trends in immunology, 2002

637 citations recorded by the source corpus.

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

Inflammatory Resolution: new opportunities for drug discovery (2004, background): “These results, and the fact that the ALXR binds other endogenous protein”
Macrophage activation by endogenous danger signals (2008, background): “These monocytes express high levels of formyl-peptide receptor 1 (FPR1), which, when ligated, suppresses the production of pro-inflammatory cytokines and removes damaged cells [106].”
International Union of Basic and Clinical Pharmacology. LXXIII. Nomenclature for the Formyl Peptide Receptor (FPR) Family (2009, background): “In this article, agonists for the FPRs are listed in separate groups in Table 5, based on their chemical constituents and origins.”
Cathelicidins, multifunctional peptides of the innate immunity (2004, background): “[98] for an overview of the FPR family), as indicated by the identification of an agonistic ligand specific for this receptor in monocytes, and by the ability of LL-37 to Figure 3.”