Current Concepts of the Pathogenesis of the Acute Coronary Syndromes
By Peter Libby. Circulation, 2001
1,647 citations recorded by the source corpus.
Subfields: Cardiology and Cardiovascular Medicine, Epidemiology, Surgery
View Paper: https://www.semanticscholar.org/paper/cbc0e781d58473a3b15553ce7a47fc8d3ad7966d
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In Cardiology and Cardiovascular Medicine
In Epidemiology
In Surgery
Citations in the corpus
- ACC/AHA 2007 guidelines for the management of patients with unstable angina/non-ST-Elevation myocardial infarction: a report of the American College of Cardiology/American Heart Association Task Force on Practice Guidelines (Writing Committee to Revise the 2002 Guidelines for the Management of Patie (2007, neutral): “Circulation 2001;104:365;(7) © 2001 Lippincott, Williams & Wilkins; The Lancet, 358, Hamm CW, Bertrand M, Braunwald E.”
- The clinical implications of endothelial dysfunction. (2003, neutral): “(82) and reverse endothelial dysfunction (64,80,83–86).”
- Management of acute coronary syndromes in patients presenting without persistent ST-segment elevation. (2002, background): “Plaques prone to rupture have a large lipid core, low smooth muscle cell density, high macrophage density, thin fibrous cap–disorganized collagen and high tissue factor concentration [15] [16] [17] [18] .”
- Androgens and cardiovascular disease. (2003, neutral): “Atherosclerosis involves interaction between the cells of the arterial wall (endothelial and smooth muscle cells) with those migrating into it (macrophages) (166).”
- Cytokines in atherosclerosis: pathogenic and regulatory pathways. (2006, neutral): “cular, and peripheral vascular beds (394).”
- Burnout and risk of cardiovascular disease: evidence, possible causal paths, and promising research directions. (2006, background): “, & Libby, P. (2002). Diabetes and athero-”
- Intelligent Image-Activated Cell Sorting. (2018, positive): “The disorder is initiated by disruption of atherosclerotic plaques, followed by further platelet activation, resulting in local occlusion or distal embolism by resultant platelet aggregates (Libby, 2001).”
- Research Progress on the Relationship between Atherosclerosis and Inflammation (2018, background): “In late-stage atherosclerosis, a large number of macrophages and other inflammatory cytokines infiltrate the vessel wall, secrete matrix metalloproteinases (MMPs), and degrade collagen fibers in the extracellular matrix of the plaque, resulting in plaque rupture, bleeding, and thrombosis [11,12].”