Neuroscience of attention-deficit/hyperactivity disorder: the search for endophenotypes

By F. X. Castellanos, Rosemary Tannock. Nature Reviews Neuroscience, 2002

1,849 citations recorded by the source corpus.

Subfields: Cognitive Neuroscience, Psychiatry and Mental health

View Paper: https://www.semanticscholar.org/paper/cb3fad347fc15a2bee343a3cad46d67839b85a1c

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Cognitive Neuroscience

In Psychiatry and Mental health

Citations in the corpus

An integrative theory of attention-deficit/ hyperactivity disorder based on the cognitive and affective neurosciences (2005, background): “On the other hand, theories remain relatively unintegrated from a cognitive and affective neuroscience point of view ~Berger & Posner, 2000; Castellanos & Tannock, 2002!.”
Differential patterns of striatal activation in young children with and without ADHD. (2003, background): “There is evidence that there are multiple etiologic pathways to ADHD (Castellanos and Tannock 2002), and subtypes diagnosed according to DSM-IV may represent different groups.”
Trajectories of brain development: point of vulnerability or window of opportunity? (2003, background): “The result of this process is an endophenotype, which refers to heritable traits that increase the risk to develop or manifest a given disease [39], and offers a new approach to elucidating the underlying mechanisms of action of common symptoms across disorders [3].”
From single to multiple deficit models of developmental disorders. (2006, neutral): “Other researchers studying the neuropsychology of ADHD are also advocating a multiple deWcit view of this disorder ( Castellanos & Tannock, 2002; Nigg, Willcutt, Doyle, & Sonuga-Barke, 2005; Sonuga-Barke, 2005).”
A dynamic developmental theory of attention-deficit/hyperactivity disorder (ADHD) predominantly hyperactive/impulsive and combined subtypes. (2005, background): “…hypofunction because the majority of findings from a variety of research fields seem to converge on dopamine in the etiology of ADHD (Biederman & Faraone 2002; Castellanos & Tannock 2002; Grace 2001; Johansen et al. 2002; Sagvolden & Sergeant 1998; Sagvolden et al. 1998; Solanto et al. 2001b).”
The Neuropsychopharmacology of Fronto-Executive Function: Monoaminergic Modulation (2009, background): “ADHD is often associated with genetic alterations in catecholamine signaling (Castellanos & Tannock 2002).”