Sex differences in pain and analgesia: the role of gonadal hormones

By R. Craft, J. Mogil, A. Aloisi. European Journal of Pain, 2004

595 citations recorded by the source corpus.

Subfields: Behavioral Neuroscience, Endocrinology, Diabetes and Metabolism, Physiology

View Paper: https://www.semanticscholar.org/paper/cab67303fc615ca67157f41dbf12dba170f452a3

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Behavioral Neuroscience

1. Irene Tracey
2. P. Mantyh

In Physiology

1. Irene Tracey
2. Roger B. Fillingim
3. P. Mantyh

Citations in the corpus

• Studying sex and gender differences in pain and analgesia: a consensus report. (2007, background): “However, later human and animal research has produced equivocal results regarding sex differences in opioid analgesia [43].”
• The organizational-activational hypothesis as the foundation for a unified theory of sexual differentiation of all mammalian tissues (2009, unknown): “For example, adult and neonatal manipulations of gonadal hormones have been found to contribute to sex-typical responses to stress and nociceptive stimuli, and sex differences in learning and cognition (Handa et al., 1994; Shors and Miesegaes, 2002; Craft et al., 2004; McCarthy and Konkle, 2005).”
• Sex, Gender, and Pain: A Review of Recent Clinical and Experimental Findings (2009, background): “oid antinociception in rodents, though the magnitude and direction of these effects can depend on multiple factors.(77,131) We have previously concluded(131) that the preclinical evidence suggests that conditions characterized by relatively high estradiol levels are associated with reduced sensitivi”
• What we know about primary dysmenorrhea today: a critical review. (2015, background): “It is, however, essential to consider menstrual cycle phase when investigating pain sensitivity in women with primary dysmenorrhea since the unique hormone environment of each menstrual phase may impact pain perception (Fillingim and Ness, 2000; Craft et al., 2004; Aloisi and Bonifazi, 2006).”
• The cerebral signature for pain perception and its modulation. (2007, background): “and Its Modulation”
• TRPA1 contributes to cold, mechanical, and chemical nociception but is not essential for hair-cell transduction. (2006, unknown): “Sex differences in response to painful stimuli and the increased sensitivity of female rodents have been well documented; these differences have been partly attributed to the influence of gonadal steroid hormones (Berkley, 1997; Fillingim and Ness, 2000; Craft et al., 2004).”