IL-17 is essential for host defense against cutaneous Staphylococcus aureus infection in mice.

By John S. Cho, E. Pietras, Nairy C Garcia, R. Ramos, David Farzam, H. Monroe, Julie E. Magorien, Andrew Blauvelt, J. Kolls, A. Cheung and 3 more. Journal of Clinical Investigation, 2010

663 citations recorded by the source corpus.

Subfields: Dermatology, Immunology, Infectious Diseases

View Paper: https://www.semanticscholar.org/paper/c8a5277b66a439be27f52ded559240b2bd14053e

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Dermatology

1. D. Cua

In Immunology

1. Victor J. Torres
2. Francis Alonzo
3. C. Tato
4. D. Cua
5. Tamara Reyes-Robles
6. R. Flavell
7. S. Gaffen

In Infectious Diseases

1. Victor J. Torres
2. Francis Alonzo
3. Tamara Reyes-Robles

Citations in the corpus

• Innate IL-17-producing cells: the sentinels of the immune system (2010, background): “aureus , whereas αβ T cell deficiency did not affect IL-17 production at this early timepoin”
• Staphylococcus aureus leukotoxin ED targets the chemokine receptors CXCR1 and CXCR2 to kill leukocytes and promote infection. (2013, background): “Indeed, quantitative and qualitative disruption of either PMNs or T cells, especially effector subsets that secrete IL-17 or IFNγ, greatly increase the susceptibility to S. aureus infection (Alonzo et al., 2012; Alonzo et al., 2013; Cho et al., 2010; Lin et al., 2009).”