Ligands for L-selectin: homing, inflammation, and beyond.
By S. Rosen. Annual Review of Immunology, 2004
800 citations recorded by the source corpus.
Subfields: Immunology, Immunology and Allergy, Oncology
View Paper: https://www.semanticscholar.org/paper/c48d8f9447bf1ad729114d76b06bf04c091fdfd0
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Citations in the corpus
- Trophoblast differentiation during embryo implantation and formation of the maternal-fetal interface. (2004, background): “larly well suited for initiating implantation because of its many special attributes (48).”
- Monocyte and macrophage heterogeneity (2005, background): “However, as anticipated by their expression of CCR2 and CD62L (molecules that are known to be involved in inflammatory-cell recruitmen”
- Endometrial receptivity markers, the journey to successful embryo implantation. (2006, background): “Sulphatases are capable of removing a sulphate moiety from natural sulphated oligosaccharides, which prevents selectin binding to its receptors (Rosen, 2004).”
- Glycosylation in cellular mechanisms of health and disease. (2006, background): “This cell adhesion system is highly regulated on specific cell surfaces including the endothelium of the vasculature and on most leukocytes, thereby contributing to leukocyte trafficking responses that are essential in immune-system homeostasis, hematopoiesis, and inflammation (Rosen, 2004).”
- Sialic acids in human health and disease (2008, neutral): “These selectin-mediated interactions are dependent on additional modifications of the glycans, such as fucosylation and sulfation, and mediate processes involved in inflammation, lymphocyte recirculation, blood coagulation and reperfusion injury [96,97].”
- Glycan-based interactions involving vertebrate sialic-acid-recognizing proteins (2007, background): “Selectins The second group of sialic-acid-binding proteins discovered was the selectins, which are involved in many cell–cell interactions in immunity, haemostasis and inflammatio”
- Immune cell migration in inflammation: present and future therapeutic targets (2005, background): “L-selectin is expressed on most circulating leukocytes and is the key receptor that initiates leukocyte capture events in high endothelial venules in secondary lymphoid tissues and at peripheral sites of injury and inflammation, which inducibly express specific sulfated and fucosylated sialoglycopro”