Nociceptors--noxious stimulus detectors.

By Clifford J. Woolf, Qiufu Ma. Neuron, 2007

993 citations recorded by the source corpus.

Subfields: Cellular and Molecular Neuroscience, Cognitive Neuroscience, Sensory Systems

View Paper: https://www.semanticscholar.org/paper/bc2c7ba11900125b7928d1205bdfb34b1eea49e4

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Cellular and Molecular Neuroscience

1. Clifford J. Woolf
2. Linda R. Watkins
3. E. Milligan
4. Jürgen Sandkühler
5. Ardem Patapoutian
6. J. Scholz
7. S. Tate
8. R. Ji

In Cognitive Neuroscience

1. Clifford J. Woolf
2. Jürgen Sandkühler
3. J. Scholz
4. Linda R. Watkins
5. G. Moalem-Taylor
6. P. Austin

In Sensory Systems

1. Clifford J. Woolf
2. Ardem Patapoutian
3. Jürgen Sandkühler
4. S. Tate
5. D. Julius

Citations in the corpus

• Neuropathic Pain (2009, background): “Nociceptive pain is an alarm mediated by highthreshold unmyelinated C or thinly myelinated Aδ primary sensory neurons that feed into nociceptive pathways of the central nervous system (CNS) (Woolf & Ma 2007).”
• Central sensitization: Implications for the diagnosis and treatment of pain (2010, background): “…the activity-dependent synaptic plasticity in the spinal cord that generates post-injury pain hypersensitivity [259], and that became termed ‘‘central sensitization” [272], as well as the current state of understanding of the cellular and molecular mechanisms responsible for this form of neuronal…”
• Transient receptor potential channels: targeting pain at the source (2009, background): “The first neural mediator of this crucial alarm system is the nociceptor”
• Models and mechanisms of hyperalgesia and allodynia. (2009, background): “Peripheral mechanisms contributing to hyperalgesia and allodynia have been reviewed extensively (194, 587).”
• Pathological and protective roles of glia in chronic pain (2009, background): “1 inset); and a central nerve terminal, which is the presynaptic portion of the synaptic relay to pain-projection neurons in the dorsal horn of the spinal cor”
• Central Sensitization: A Generator of Pain Hypersensitivity by Central Neural Plasticity (2009, neutral): “nociceptive pathways by peripheral stimuli of sufficient intensity to lead to or to threaten tissue damage (noxious stimuli).(374) Nociception, the detection of noxious stimuli,(282) is a protective process that helps prevent”
• Estrogen Rapidly Enhances Incisional Pain of Ovariectomized Rats Primarily through the G Protein-Coupled Estrogen Receptor (2014, background): “Administration of estrogen has been shown to result in sensitization of nociceptive neurons, resulting in a decrease of the pain threshold [7,8].”