Inflammatory cytokines and postmyocardial infarction remodeling.
By M. Nian, Paul Lee, N. Khaper, Peter. P. Liu. Circulation Research, 2004
1,048 citations recorded by the source corpus.
Subfields: Cardiology and Cardiovascular Medicine, Molecular Biology, Pathology and Forensic Medicine
View Paper: https://www.semanticscholar.org/paper/ae6dfbfd507db401154481a3d8e431283567d8ec
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In Cardiology and Cardiovascular Medicine
In Molecular Biology
In Pathology and Forensic Medicine
Citations in the corpus
- The Biological Basis for Cardiac Repair After Myocardial Infarction: From Inflammation to Fibrosis. (2016, background): “Nonetheless, despite preclinical data suggesting that cytokines and chemokines serve to aggravate ischemic injury and remodeling, it should be recognized that several studies conflict with this paradigm, showing that these mediators engender cardioprotective responses and pleiotropic cellular effect”
- Myocardial matrix remodeling and the matrix metalloproteinases: influence on cardiac form and function. (2007, background): “First, the initial myocardial injury is associated with an acute inflammatory response and the release of degradative enzymes that facilitate wound healing (43, 303, 491).”
- Mitogen-activated protein kinase signaling in the heart: angels versus demons in a heart-breaking tale. (2010, background): “Acting in a paracrine fashion, they play a large role in myocardial remodeling (300).”