Inflammatory links between obesity and metabolic disease.

By C. Lumeng, Alan R. Saltiel. Journal of Clinical Investigation, 2011

2,220 citations recorded by the source corpus.

Subfields: Endocrine and Autonomic Systems, Epidemiology, Physiology

View Paper: https://www.semanticscholar.org/paper/a731483c3a6ca605d7ea1a77fb5cd3d1db005430

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Endocrine and Autonomic Systems

1. Byung-Cheol Lee
2. B. Kahn
3. Jongsoon Lee

In Epidemiology

1. Jamal Naderi
2. M. Longo
3. Byung-Cheol Lee
4. C. Miele
5. Jongsoon Lee
6. G. A. Raciti
7. F. Beguinot
8. Federica Zatterale

In Physiology

1. Jamal Naderi
2. M. Longo
3. Byung-Cheol Lee
4. C. Miele
5. Jeremy K. Nicholson
6. B. Kahn
7. Jongsoon Lee
8. G. A. Raciti

Citations in the corpus

• Metabolic syndrome update. (2016, neutral): “grade, systemic inflammation [55,56].”
• Discovery of a class of endogenous mammalian lipids with anti-diabetic and anti-inflammatory effects (2014, background): “Innate immunity is activated in WAT in obesity and may contribute to the insulin-resistant state (Lumeng and Saltiel, 2011).”
• Metabolic dysfunction drives a mechanistically distinct proinflammatory phenotype in adipose tissue macrophages. (2014, background): “Macrophages accumulate in adipose tissue of obese mice and humans (Weisberg et al., 2003; Xu et al., 2003) and are key contributors to inflammation and obesity-induced insulin resistance (Chawla et al., 2011; Lumeng and Saltiel, 2011; Olefsky and Glass, 2010; Wellen and Hotamisligil, 2005).”
• Impact of the gut microbiota on inflammation, obesity, and metabolic disease (2016, background): “This production promotes a chronic, low-grade inflammatory status, induces the recruitment and activation of many mature immune cells (including mast cells, macrophages, and dendritic cells) in metabolic tissues and particularly in adipose tissues, and also induces recruitment and activation of othe”
• Differential Macrophage Programming in the Tumor Microenvironment (2012, neutral): “In this context, tissue-resident macrophages assist with combating infection [1, 2], resolving acute inflammation [3], and in regulating metabolic response to tissue stress [4, 5].”
• INFLAMMATION AND LIPID SIGNALING IN THE ETIOLOGY OF INSULIN RESISTANCE (2012, neutral): “…resistance has been intensely studied (Qatanani and Lazar, 2007; Shulman, 2000), and it is now recognized that chronic tissue inflammation is an important cause of obesity-induced insulin resistance (Attie and Scherer, 2009; Lumeng and Saltiel, 2011; Olefsky and Glass, 2010; Shoelson et al., 2007).”