Signaling pathways in skeletal muscle remodeling.

By R. Bassel-Duby, E. Olson. Annual Review of Biochemistry, 2006

843 citations recorded by the source corpus.

Subfields: Genetics, Molecular Biology, Physiology

View Paper: https://www.semanticscholar.org/paper/a4e924fc14329aa40a5d78194d2fb52f0d70f59c

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Citations in the corpus

Integrative biology of exercise. (2014, neutral): “Comprehensive discussion of skeletal muscle fiber types and the gene programs responsible for fiber-specific properties are beyond the scope of this Review and have been summarized elsewhere (Bassel-Duby and Olson 2006; Saltin et al., 1977; Schiaffino and Reggiani 1996; Zierath andHawley 2004).”
AMPK and PPARd Agonists Are Exercise Mimetics (2008, positive): “Although not all genes regulated by either exercise (data not shown) or exercise-PPARd interaction (nonoverlapping signature, Figure 4D) are AMPK dependent, two key findings assign a critical role for the kinase in promoting endurance compared to other known exercise signals (Bassel-Duby and Olson, ”
Transcriptional mechanisms regulating skeletal muscle differentiation, growth and homeostasis (2011, background): “During postnatal adaptation of muscle, or muscle remodelling, aspects of early developmental programmes can be reactivated and can cooperate with specific factors to determine fibre-type characteristic”
The Molecular Bases of Training Adaptation (2007, background): “Skeletal muscle atrophy is characterised by a decrease in structural and contractile protein content and fibre diameter (Jackman and Kandarian 2004; Bassel-Duby and Olson 2006).”
A Micropeptide Encoded by a Putative Long Non-coding RNA Regulates Muscle Performance (2015, neutral): “Ca controls the normal function of striated muscle by acting as theprimary regulator of the sarcomeric contractilemachinery and as a second messenger in the signal transduction pathways that control muscle growth, metabolism, and pathological remodeling (Bassel-Duby and Olson, 2006; Berchtold et al.”
Fiber types in mammalian skeletal muscles. (2011, background): “Many other reviews in the last few years have covered various aspects of this subject, and the readers are referred also to them (42, 59, 489, 605, 704, 715, 755).”
Regulation of energy metabolism by long-chain fatty acids. (2014, background): “Likewise, overexpression a constitutively active form of PPARd in skeletal muscle resulted in mitochondrial biogenesis and a shift of the fiber type from fast twitch to oxidative slow twitch [132], a response of muscle similar to long-term endurance training [125], whereas a PPARd agonist was unable”
A family of microRNAs encoded by myosin genes governs myosin expression and muscle performance. (2009, background): “The activation of slow myofiber genes in skeletal muscle depends on calcium signaling through calcineurin and various kinases (Bassel-Duby and Olson, 2006; Wu et al., 2000), leading us to propose that b-MHC and Myh7b are activated independently through such signals, resulting in the production of…”
Butyrate Improves Insulin Sensitivity and Increases Energy Expenditure in Mice (2009, background): “The types I fibers are distinct from the type II fibers in several properties (25).”
The many roles of histone deacetylases in development and physiology: implications for disease and therapy (2009, background): “Skeletal muscle fibres differ in their contractile and metabolic properties, which reflect different patterns of gene expressio”
Satellite Cells and Skeletal Muscle Regeneration (2015, positive): “The Akt-1/mTOR pathway induces various trophic effects via several pathways (18). mTOR activation stimulates protein synthesis by phosphorylating p70 S6k that activates the ribosomal protein S6. mTOR also exerts anabolic effects by inhibiting the translation repressor 4E-protein binding 1 (4E-BP1).”