The Molecular Bases of Training Adaptation

By Vernon G. Coffey, J. Hawley. Sports Medicine, 2007

758 citations recorded by the source corpus.

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

Integrative biology of exercise. (2014, positive): “IGF-1 transmits signaling along the PI3K/Akt pathway (Figure 4), resulting in the parallel activation of the mTOR pathway, producing a multitude of responses, including mRNA translation, ribosomal biogenesis, and nutrient metabolism (Coffey and Hawley 2007).”
Acute exercise remodels promoter methylation in human skeletal muscle. (2012, background): “(Coffey and Hawley, 2007).”
High-Intensity Interval Training, Solutions to the Programming Puzzle (2013, neutral): “Quantifying the neuromuscular load of an HIT session is important, since it (1) affects the HIT performance, and subsequently, the T@ _ VO2max (see Part I [1]); (2) might have potential carry-over effects for subsequent training sessions [99–101]; (3) might modulate long-term neuromuscular adaptatio”
Physiological adaptations to interval training and the role of exercise intensity (2017, background): “Exercise is traditionally defined as either endurance or strength or viewed as a continuum anchored by these common descriptors (Coffey & Hawley, 2007; Hawley et al. 2014).”
Exercise metabolism and the molecular regulation of skeletal muscle adaptation. (2013, background): “Over time, skeletal muscle demonstrates remarkable malleability in functional adaptation and remodeling in response to contractile activity (Flück and Hoppeler, 2003; Coffey and Hawley, 2007).”