BACH1, a novel helicase-like protein, interacts directly with BRCA1 and contributes to its DNA repair function.

By S. Cantor, D. Bell, Shridar Ganesan, Elizabeth M. Kass, R. Drapkin, S. Grossman, D. Wahrer, D. Sgroi, William Arbuthnot Sir Lane, D. Haber and 1 more. Cell, 2001

710 citations recorded by the source corpus.

Subfields: Genetics, Molecular Biology

View Paper: https://www.semanticscholar.org/paper/a056ebca6b1b67faa5b6fc9f38c27c4db4e68563

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Citations in the corpus

Homologous recombination and human health: the roles of BRCA1, BRCA2 and associated proteins (2015, background): “…predisposition, such that BRIP1 is considered to be a Fanconi anemia gene subtype (FA-J); monoallelic mutations are associated with breast and ovarian cancer predisposition (Cantor et al. 2001; Levitus et al. 2005; Levran et al. 2005; Litman et al. 2005; Seal et al. 2006; Rafnar et al. 2011).”
Discovery of cell-type specific DNA motif grammar in cis-regulatory elements using random Forest (2018, neutral): “, JDP2 [54], BACH1 [55] and NFE2 [56, 57]).”
Molecular views of recombination proteins and their control (2003, background): “pl?O14867">BACH1 , which is a putative DNA helicas”
How the fanconi anemia pathway guards the genome. (2009, background): “In the absence of BRCA1, reduced FANCD2 ubiquitination and focus formation are observed, suggesting an essential function for this protein in FA pathway activation.”
Mitotic homologous recombination maintains genomic stability and suppresses tumorigenesis (2010, neutral): “BRIP1, a DNA helicase originally identified as BACH1 and later as FANCJ, binds to the BRCA1 BRCT repeats in S phase following its phosphorylatio”