Inflammation and cancer

By Lisa M. Coussens, Zena Werb. Nature, 2002

7,865 citations recorded by the source corpus.

Subfields: Immunology, Oncology

View Paper: https://www.semanticscholar.org/paper/94bdcdb2545b006288494f048f749028ba8b8a74

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Immunology

1. A. Mantovani
2. A. Sica
3. P. Allavena
4. M. Karin
5. J. Pollard
6. D. Hanahan
7. J. Joyce
8. Zena Werb

In Oncology

1. M. Karin
2. A. Mantovani
3. D. Hanahan
4. Zena Werb
5. J. Pollard
6. P. Allavena
7. A. Sica
8. J. Joyce

Citations in the corpus

• Psychological Stress in Childhood and Susceptibility to the Chronic Diseases of Aging: Moving Towards a Model of Behavioral and Biological Mechanisms (2011, background): “It helps the tumor cell divide rapidly, gain access to a blood supply, subvert detection by the immune system, and migrate to new tissues (Coussens & Werb, 2002; Mantovani, Allavena, Sica, & Balkwill, 2008).”
• Cancer-related inflammation, the seventh hallmark of cancer: links to genetic instability. (2009, background): “Recent years have seen a renaissance of the inflammationcancer connection stemming from different lines of work and leading to a generally accepted paradigm [1-4].”
• The role of IL-6 and STAT3 in inflammation and cancer. (2005, background): “However, recent evidence suggests that the immune system more closely resembles a ‘‘double edged sword,’’ rather than a benign protector [1].”
• IL-6 in inflammation, autoimmunity and cancer (2020, background): “Chronic inflammation is often accompanied with auto-immune disease and cancer (2, 4, 9).”
• The Tumor Microenvironment: A Milieu Hindering and Obstructing Antitumor Immune Responses (2020, background): “In fact, the association of chronic inflammation with tumor progression has been shown a long time ago (26).”
• Macrophages, innate immunity and cancer: balance, tolerance, and diversity. (2010, background): “Introduction After a long eclipse, in recent years different lines of work have lead to a renaissance of the inflammation–cancer connection [1–3].”