Association pathways of the prefrontal cortex and functional observations

By Michael Petrides, Deepak N. Pandya. 2002

213 citations recorded by the source corpus.

Subfields: Artificial Intelligence, Cognitive Neuroscience

View Paper: https://www.semanticscholar.org/paper/8d1ead0fe3d9340d9fbe80fb1cfc4251ca5e5e32

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Citations in the corpus

Investigations into resting-state connectivity using independent component analysis (2005, unknown): “…lesions affecting the lateral posterior parietal areas lead to severe deficits in spatial attention (Mesulam 1981; Posner et al. 1984), while disrupting the input to the region prevents patients from benefiting from spatial cues (Alivisatos & Milner 1989; Koski et al. 1998; Petrides & Pandya 2002).”
Consciousness without a cerebral cortex: A challenge for neuroscience and medicine (2007, background): “Failure to notice the relevance of prefrontotectal pathways abounds even in authoritative reviews of prefrontal anatomy (Petrides & Pandya 2002), and consequently, the putative functions of such connectivity have been overlooked or downplayed by recent models about the neural control of human…”
Segmentation of subcomponents within the superior longitudinal fascicle in humans: a quantitative, in vivo, DT-MRI study. (2005, background): “In monkeys, SLF III connects the rostral part of the inferior parietal lobule with the lateral inferior frontal lobe in a bidirectional way (Preuss and Goldman-Rakic, 1989; Petrides and Pandya, 2002).”
Left ventrolateral prefrontal cortex and the cognitive control of memory. (2007, background): “This atter subdivision maps to what Petrides and Pandya (2002b) erm area 47/12, noting the homology between area 47 in the uman and area 12 in the macaque monkey (Fig.”
Breakdown of functional connectivity in frontoparietal networks underlies behavioral deficits in spatial neglect. (2007, unknown): “The AF connects middle frontal areas with superior temporal areas (Petrides and Pandya, 2002), providing a possible structural basis for the disruption of MFG-STS FC.”
Cortex and Memory: Emergence of a New Paradigm (2009, background): “Most all pathways between frontal and posterior association cortices are bidirectional, and so are the connections between those cortices and thalamic nuclei (Petrides & Pandya, 2002; review in Fuster, 2008).”
The self-regulating brain: Cortical-subcortical feedback and the development of intelligent action (2007, background): “It may therefore play a mediating role between the HPC and DLPFC, initiating the retrieval of episodic information and maintaining its activation in working memory (Mesulam, 2002; Petrides & Pandya, 2002).”
Cognitive processing speed and the structure of white matter pathways: Convergent evidence from normal variation and lesion studies (2008, unknown): “…(3) prefrontal cortex of the contralateral hemisphere through transcallosal fiber tracts; and (4) the basal ganglia through fronto-striatal projections (Alexander et al., 1990; Burgel et al., 2006; Lehericy et al., 2004; Mori et al., 2002; Petrides and Pandya, 2002; Schmahmann and Pandya, 2006).”
The case for the development and use of “ecologically valid” measures of executive function in experimental and clinical neuropsychology (2006, background): “For example, Petrides (1994, 1998; Petrides & Pandya, 2002) maintains that the midventrolateral PFC " subserves the expression within memory of various first-order executive processes, such as (the) active selection, comparison and judgement of stimuli, " (p. 107) without specifying further what…”
DTI measures in crossing-fibre areas: Increased diffusion anisotropy reveals early white matter alteration in MCI and mild Alzheimer's disease (2011, background): “Damage to the SLF II is known to result in disorders of spatial working memory (Preuss and Goldman-Rakic, 1989; Petrides and Pandya, 2002).”
Bridging emotion theory and neurobiology through dynamic systems modeling (2005, background): “…pathways between the HPC and OFC/DLPFC (Ongur & Price 2000), which may support the value-related modulation of retrieval and working memory (Petrides & Pandya 2002); (2) bidirectional pathways between the HPC and ACC, extending to the DLPFC, necessary for selective attention and monitoring…”