NETs: a new strategy for using old weapons.

By V. Papayannopoulos, Arturo Zychlinsky. Trends in immunology, 2009

676 citations recorded by the source corpus.

Subfields: Immunology, Molecular Biology

View Paper: https://www.semanticscholar.org/paper/8beb1706ec9f969e0cc4b70629213d14330243eb

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Immunology

1. P. Kubes
2. Arturo Zychlinsky
3. V. Papayannopoulos
4. Kathleen D. Metzler
5. B. Yipp
6. M. Otto
7. Abdul Hakkim
8. C. Lowell

In Molecular Biology

1. M. Otto
2. P. Kubes
3. Christian Weber
4. V. Papayannopoulos
5. Arturo Zychlinsky
6. Kathleen D. Metzler
7. H. Noels

Citations in the corpus

• The Multifaceted Functions of Neutrophils (2013, background): “Moreover, histones themselves have significant antimicrobial activity (124).”
• Neutrophil elastase and myeloperoxidase regulate the formation of neutrophil extracellular traps (2010, background): “NETs are implicated in immune defense, sepsis, and autoimmunity (Clark et al., 2007; Kessenbrock et al., 2009; Papayannopoulos and Zychlinsky, 2009; Hakkim et al., 2010).”
• Intravascular neutrophil extracellular traps capture bacteria from the bloodstream during sepsis. (2012, background): “Interestingly, neutrophils are endowed with effector mechanisms termed neutrophil extracellular traps (NETs, large webs of decondensed chromatin coated with granule proteins) that are expelled from activated neutrophils that can capture and kill microbes in the extracellular space (Brinkmann et al.,”
• Atherosclerosis: current pathogenesis and therapeutic options (2011, background): “Furthermore, neutrophils activated through TLRs, Fc receptors or cytokine receptors can release nuclear content (DNA fibers, chromatin) that forms a scaffold containing antimicrobial proteins, such as LL-37, termed neutrophil extracellular traps (NETs”
• Dying for a cause: NETosis, mechanisms behind an antimicrobial cell death modality (2011, neutral): “However, only very few NETs, if any, are generated by HL-60 cells, regardless of their differentiation state.(30,41,50) This may be explained by defects in their ability to induce autophagy.”
• Pathogenicity and virulence of Staphylococcus aureus (2021, background): “(“NETosis”) [136], is also believed to contribute to host defense against S.”