Intravascular neutrophil extracellular traps capture bacteria from the bloodstream during sepsis.

By B. McDonald, R. Urrutia, B. Yipp, C. Jenne, P. Kubes. Cell Host and Microbe, 2012

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Neutrophils: Between Host Defence, Immune Modulation, and Tissue Injury (2015, background): “aureus infection [45] and dynamic and intravascular NET formation in bacterial [46] and viral infections [47].”
Thrombosis: tangled up in NETs. (2014, background): “There could be a benefit of intravascular NET formation in septic conditions where containing an overwhelming bacteremia is likely protective for the host.(23,29) The large quantities of antimicrobial toxic products released with NETs, in particular histones, the main protein component of NETs,(2) c”
Neutrophil extracellular traps sequester circulating tumor cells and promote metastasis. (2013, neutral): “DNAse is thought to act via degradation of the DNA structure of NETs (48).”
Platelets at the interface of thrombosis, inflammation, and cancer. (2015, background): “The resultant web-like networks of chromatin, histones, and degradative enzymes known as neutrophil extracellular traps (NETs) capture circulating microbes and thus limit their ability to colonize alternate sites.(35,36) Both in vitro and in vivo studies have illustrated that activated platelets adh”
Platelets in Inflammation and Atherogenesis (2015, background): “Platelet TLR-4 (74) as well as platelet β-defensins have been implicated in NET formation (75, 76).”