Atherosclerosis: current pathogenesis and therapeutic options

By Christian Weber, H. Noels. Nature Medicine, 2011

2,166 citations recorded by the source corpus.

Subfields: Immunology, Rheumatology, Surgery

View Paper: https://www.semanticscholar.org/paper/0009553946f3eaab07e59ca7039b7e4ea08d66b5

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Immunology

1. Peter Libby
2. G. Hansson
3. A. Lichtman
4. Alberico L. Catapano
5. J. Bentzon
6. P. Ridker
7. I. Tabas
8. V. Z. Rocha

In Rheumatology

1. G. Hansson
2. Peter Libby
3. I. Graham
4. Børge G. Nordestgaard

In Surgery

1. A. Lichtman
2. Peter Libby
3. I. Graham
4. G. Hansson
5. Jan Boren
6. M. Chapman
7. Marja-Riitta Taskinen
8. Alberico L. Catapano

Citations in the corpus

• Medical hyperspectral imaging: a review (2014, background): “Coronary artery disease is a leading cause of death and morbidity worldwide.(175) It arises from atherosclerosis through a slowly progressing lesion formation and luminal narrowing of arteries.”
• Atherosclerosis: Process, Indicators, Risk Factors and New Hopes (2014, background): “There are a couple of valuable review papers addressing the pathogenesis of atherosclerosis.([7,8]) In this study, we aimed to review the mechanism of atherosclerosis and its risk factors, focusing on new findings in atherosclerosis markers and its risk factors.”
• Immune Effector Mechanisms Implicated in Atherosclerosis: From Mice to Humans (2013, background): “Local cellular responses prompt expression of leukocyte adhesion molecules on the endothelial lining of the artery, as well as expression of chemokines by the vascular cells (Mestas and Ley, 2008; Weber and Noels, 2011).”
• Akkermansia Muciniphila Protects Against Atherosclerosis by Preventing Metabolic Endotoxemia-Induced Inflammation in Apoe −/− Mice (2016, background): “Inflammation and hypercholesterolemia are the 2 key etiological factors for atherosclerosis.(10,39) In the initial stage of atherosclerosis, injured or inflamed endothelium secretes adhesion molecules and chemokines to facilitate the recruitment and transmigration of leukocytes into the intima.”
• Low-density lipoproteins cause atherosclerotic cardiovascular disease: pathophysiological, genetic, and therapeutic insights: a consensus statement from the European Atherosclerosis Society Consensus Panel (2020, background): “to up-regulate adhesion molecules and chemokines that trigger the recruitment of monocytes—typically inflammatory Ly6C monocytes—into the artery wall.(131) The importance of oxidized phospho-”