Para-inflammation in the aging retina.

By Heping Xu, Mei Chen, John V. Forrester. Progress in retinal and eye research, 2009

657 citations recorded by the source corpus.

Subfields: Immunology, Neurology, Ophthalmology

View Paper: https://www.semanticscholar.org/paper/82b58a1d6a4c78ee724c0c9107c889bc35f10130

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Immunology

1. Antero Salminen
2. I. Bhutto
3. G. Lutty
4. K. Kaarniranta

In Ophthalmology

1. I. Bhutto
2. G. Lutty

Citations in the corpus

• Molecular basis of the inner blood-retinal barrier and its breakdown in diabetic macular edema and other pathological conditions. (2013, background): “disease” (Adamis and Berman, 2008) or has been described as a response to “para-inflammation” (Xu et al., 2009).”
• Oxidative stress and diabetic retinopathy: Molecular mechanisms, pathogenetic role and therapeutic implications (2020, background): “response to remove oxidation products by phagocytosis [232].”
• Inflammation in Diabetic Retinopathy (2011, neutral): “Aging or hypertension in the absence of diabetes increase expression of proinflammatory molecules in the retina (Silva et al., 2007; Xu et al., 2009), yet these conditions do not cause a diabetic-like retinopathy.”
• Inflammation and its role in age-related macular degeneration (2016, background): “the maintenance of tissue homeostasis and the clearance of debris from the subretinal space [158, 228, 229].”
• Understanding age-related macular degeneration (AMD): Relationships between the photoreceptor/retinal pigment epithelium/Bruch’s membrane/choriocapillaris complex (2012, background): “3.2 Other changes in innate immunity during AMD Forrester et al suggested that chronic para-inflammation contributes to the initiation of AMD (Xu et al., 2009).”
• Diabetic retinopathy: current understanding, mechanisms, and treatment strategies. (2017, background): “While this response promotes homeostasis and normal tissue repair, in the short-term, chronic parainflammation contributes to initiation and progression of multiple disease processes (89).”