Role of Genotype in the Cycle of Violence in Maltreated Children

By Avshalom Caspi, J. McClay, Terrie E. Moffitt, Jonathan Mill, Judy L. Martin, I. Craig, Alan H. Taylor, Richie Poulton. Science, 2002

4,456 citations recorded by the source corpus.

Subfields: Behavioral Neuroscience, Clinical Psychology

View Paper: https://www.semanticscholar.org/paper/7011bc7587a8b4e209a4e8591e8878f4a3fae3ec

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Citations in the corpus

The family check-up with high-risk indigent families: preventing problem behavior by increasing parents' positive behavior support in early childhood. (2008, background): “Specifically, we know that among children assessed to be at genetic risk for problem behavior, punitive and harsh parenting practices greatly exacerbate that risk (Caspi et al., 2002).”
The Cycle of Violence Behind Bars: Traumatization and Institutional Misconduct Among Juvenile Delinquents in Confinement (2010, background): “In a landmark study, Caspi et al. (2002) added clarity to the cycle of violence by showing that those with the low-activity polymorphism in the gene encoding the neurotransmitter-metabolizing enzyme monoamine oxidase A (MAOA) and who were maltreated were likely to develop antisocial behavior.”
Gene-environment interplay and psychopathology: multiple varieties but real effects. (2006, methodology): “The study extended the Caspi et al. (2002) analysis by showing that the G · E could not be accounted for by either a passive or an evocative gene–environment correlation.”
Mediators and mechanisms of change in psychotherapy research. (2007, background): “Abused children with a genetic polymorphism (related to the metabolism of serotonin) have much higher rates of antisocial behaviors than those without this polymorphism (Caspi et al. 2002).”
Does the Effect of Impulsivity on Delinquency Vary by Level of Neighborhood Disadvantage? (2006, background): “This work has provided evidence that the gene is polymorphic (Caspi et al., 2002).”
The genetics of quantitative traits: challenges and prospects (2009, background): “A promoter variant of the monoamine oxidase A ( MAOA ) gene is associated with violent behaviour only if the individual was abused as a chil”
Genetic and environmental influences on human psychological differences. (2003, background): “Consistent with this proposition, Caspi et al. (2002) recently reported that a polymorphism associated with low activity of the enzyme monoamine oxidase A (MAOA) was associated with increased antisocial behavior among boys who had been maltreated in childhood but not among boys who had not experienc”
Beyond diathesis stress: differential susceptibility to environmental influences. (2009, background): “…the low-activity MAOA allele to antisocial behavior and another linking abuse and neglect in childhood to the same developmental outcome, led Caspi et al. (2002) to hypothesize that inconsistency in findings in both literatures could be a result of the fact that maltreatment effects are…”
Psychological Stress in Childhood and Susceptibility to the Chronic Diseases of Aging: Moving Towards a Model of Behavioral and Biological Mechanisms (2011, background): “There is also growing evidence that such variation explains variability in how early stress affects mental health (Belsky & Pluess, 2009; Binder et al., 2008; R. G. Bradley et al., 2008; Caspi et al., 2002; Gillespie, Phifer, Bradley, & Ressler, 2009).”
The evolution of transcriptional regulation in eukaryotes. (2003, background): “…al. 1996; Sinha and Kellogg 1996; Stockhaus et al. 1997; Segal, Barnett, and Crawford 1999; Lerman et al. 2003); (3) behavior (Trefilov et al. 2000; Caspi et al. 2002; Enard et al. 2002 b ; Fang, Takahashi, and Wu 2002; Hariri et al. 2002; Saito et al. 2002); (4) disease susceptibility…”
The Development of Physical Aggression from Toddlerhood to Pre-Adolescence: A Nation Wide Longitudinal Study of Canadian Children (2006, background): “Genetically informative studies such as twin, adoption, and molecular genetic studies are needed to test the contribution of genetic factors and the likely gene-environment interactions and geneenvironment correlations (e.g., Arseneault et al., 2003; Caspi et al., 2002; Dionne et al., 2003; Weaver e”