Modality-specific retrograde amnesia of fear.
By J. J. Kim, M. Fanselow. 1992
How researchers see this paper
The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.
- J. Snyder
- Fred H. Gage
- Joseph E LeDoux
- R. Duman
- Bruce S. McEwen
- R. Hen
- James Bradley Aimone
- Wei Deng
Citations in the corpus
- Reactivation of Hippocampal Cell Assemblies: Effects of Behavioral State, Experience, and EEG Dynamics (1999, background): “…role in the initial encoding of a memory but that, with time, the memory becomes independent of the hippocampus (Scoville and Milner, 1957; McGaugh and Herz, 1972; MacKinnon and Squire, 1989; Kim and Fanselow, 1992; for review, see Zola-Morgan and Squire, 1993; but see Nadel and Moscovitch, 1997).”
- Inhibition of neurogenesis interferes with hippocampus‐dependent memory function (2006, background): “The pattern to note in these results is that the cognitive processes affected by irradiation are also disrupted by lesions to the hippocampus , while those processes that remained intact are considered to be independent of hippocampal control (Kim and Fanselow, 1992; Winocur, 1991, 1992).”
- Allostasis and Allostatic Load: Implications for Neuropsychopharmacology (2000, background): “The hippocampus is important in declarative, spatial, and contextual memory and plays a role in processing of the contextual associations of strong emotions (Eichenbaum 1997; Gray 1982; Kim and Fanselow 1992; Phillips and LeDoux 1992; Pugh et al. 1997).”
- Contextual and auditory fear conditioning are mediated by the lateral, basal, and central amygdaloid nuclei in rats. (2001, background): “In contrast, contextual stimuli are processed in the hippocampus (Kim and Fanselow 1992; Phillips and LeDoux 1992; Maren et al. 1997, 1998), and hippocampal afferents to the amygdala synapse primarily on BL and BM neurons (Canteras and Swanson 1992; Maren and Fanselow 1995).”
- Memory Reconsolidation and Extinction Have Distinct Temporal and Biochemical Signatures (2004, background): “Whereas initial encoding and storage of fear memories depend on the hippocampus , permanent storage is proposed to depend on the neocortex ( Kim and Fanselow , 1992 ; Eichenbaum et al .”
- Hippocampal CA3 Output is Crucial for Ripple-Associated Reactivation and Consolidation of Memory (2009, unknown): “…no significant reduction of the SC input up to 2 weeks after Dox withdrawal (Nakashiba et al., 2008), the mutant mice treated this way have had a total of 5 weeks with an intact SC input following the training, a period sufficient for a full consolidation of recent memory (Kim and Fanselow, 1992).”
- Electroconvulsive seizure restores neurogenesis and hippocampus‐dependent fear memory after disruption by irradiation (2008, methodology): “For context testing, rats were placed back in the same boxes, cleaned with 1% acetic acid, and freezing behavior was scored every 8 s for 8 min (Kim & Fanselow, 1992).”
- Loss of Ensemble Segregation in Dentate Gyrus, but not in Somatosensory Cortex, during Contextual Fear Memory Generalization (2016, background): “Contextual fear memory retrieval shortly after conditioning depends on the hippocampus, and becomes independent of the hippocampus at a remote time point (Kim and Fanselow, 1992; Frankland and Bontempi, 2005; also see Goshen et al., 2011).”
- Systems consolidation and hippocampus: two views (2007, background): “, [28]) and socially acquired taste preferences (e.”
- Sleep Is for Forgetting (2017, unknown): “The time course over which place cells switched from the novel peak firing pattern in REM sleep to the familiar trough firing pattern corresponded with the timescale of memory con- solidation from the hippocampus to storage places outside the hippocampus as described by Kim and Fanselow (1992).”