Addiction, Dopamine, and the Review Molecular Mechanisms of Memory

By Joshua D. Berke, S. Hyman. 2000

1,088 citations recorded by the source corpus.

View Paper: https://www.semanticscholar.org/paper/68b70baec6dc862a527d25e442cc3d99078c5da7

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

Citations in the corpus

Neural mechanisms of addiction: the role of reward-related learning and memory. (2006, background): “As with other forms of memory, it is hypothesized that changes in gene expression or protein translation play an important role in memory storage (Berke & Hyman 2000, Nestler 2001).”
Adult neurogenesis: from precursors to network and physiology. (2005, background): “These results suggest that memory traces of the association between a specific context and the availability of the drug could be stored in the HF, and the hypothesis that drug addiction is an aberrant form of learning mediated by maladaptive recruitments of hippocampal-dependent memory system has re”
The reinstatement model of drug relapse: history, methodology and major findings (2003, background): “2001), or that drug addiction is an aberrant form of learning, mediated by maladaptive recruitment of certain memory systems in the brain (Di Chiara 1999; Berke and Hyman 2000; Lu et al. 2000b).”
Synaptic and Behavioral Profile of Multiple Glutamatergic Inputs to the Nucleus Accumbens (2012, background): “A prominent idea is that the glutamate input to the NAc encodes the context, cues, and descriptive features that characterize any given moment in time (Berke and Hyman, 2000; Everitt and Wolf, 2002; Kelley, 2004; Pennartz et al., 2011).”
Single cocaine exposure in vivo induces long-term potentiation in dopamine neurons (2001, background): “jpg" NDATA ITEM> ]> How do drugs of abuse modify neural circuitry and thereby lead to addictive behaviour? As for many forms of experience-dependent plasticity, modifications in glutamatergic synaptic transmission have been suggested to be particularly importan”
Opposing Patterns of Signaling Activation in Dopamine D1 and D2 Receptor-Expressing Striatal Neurons in Response to Cocaine and Haloperidol (2008, background): “…cocaine, transient induction in the striatum of IEGs such as c-fos and zif268 (also termed egr-1, krox24, or NGFI-A) has been proposed to play a role in long-term neuronal alterations (Graybiel et al., 1990; Rosen et al., 1994; Moratalla et al., 1996; Berke and Hyman, 2000; Valjent et al., 2006b).”
Flies, genes, and learning. (2001, background): “FLIES ON DRUGS Genes that affect learning are also involved in the response of flies and mammals to drugs of human abuse (Berke & Hyman 2000).”
Addiction and the brain: The neurobiology of compulsion and its persistence (2001, background): “and the hypothesis that addictive drugs produce long-lived alterations in behaviour largely by usurping normal mechanisms of associative memor”
Addiction: a disease of learning and memory. (2005, background): “This stored information must provide internal representations of the reward-related stimulus, its valuation, and a series of action sequences so that the cue can trigger an effective and efficient behavioral response (19).”
The incentive sensitization theory of addiction: some current issues (2008, background): “R habits confer compulsivity to behaviour (Tiffany 1990; Berke & Hyman 2000; Everitt et al. 2001; Hyman et al. 2006).”
The primate basal ganglia: parallel and integrative networks. (2003, background): “…with the development of reward-based learning, the acquisition of newly acquired behaviors, and plasticity (Ljungberg et al., 1991; Anglade et al., 1996; Hollerman and Schultz, 1998; Schultz, 1998; Bonci and Malenka, 1999; Matsumoto et al., 1999; Berke and Hyman, 2000; Song and Haber, 2000).”