Postinfectious irritable bowel syndrome.
By A. Dupont. Clinical Infectious Diseases, 2008
706 citations recorded by the source corpus.
Subfields: Epidemiology, Gastroenterology, Pharmacy
View Paper: https://www.semanticscholar.org/paper/67918103da3f712892718b2c6bc1ebb396f26f75
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Citations in the corpus
- Probiotics and the gut microbiota in intestinal health and disease (2010, background): “coli O157:H7 [EHEC] infections) are categorized as having postinfectious IBS”
- Gut feelings: the emerging biology of gut–brain communication (2011, background): “Surprisingly, abdominal pain or persistent visceral hyperalgesia to experimental stimuli is not a prominent feature in most patients with an uncomplicated IBD, and only a small percentage of patients with IBD or with infectious gastroenteritis, experience persistent pain once the inflammation has re”
- Intestinal microbiota in functional bowel disorders: a Rome foundation report (2012, background): “For instance, many subjects with irritable bowel syndrome (IBS) report symptom onset following an enteric infection.(22) There are also studies reporting positive effects of treatments directed at gut microbiota in patients with FBD.”
- Efficacy of an Encapsulated Probiotic Bifidobacterium infantis 35624 in Women with Irritable Bowel Syndrome (2006, background): “Another more recent development in the understanding of the pathogenesis of IBS is the recognition that in a proportion of patients, there might be an inflammatory component (7).”
- Brain–Gut–Microbe Communication in Health and Disease (2011, background): “, 2006, 2007) and prospective studies showing that the risk of developing PI-IBS is in the order of 3–36% following an enteric infection with the precise incidence depending on the infecting organism (Spiller and Garsed, 2009).”
- Gut microbiota in health and disease. (2010, background): “Irritable bowel syndrome (IBS), a disorder of the GIT with complex etiology, sometimes develops following recovery from enteric infections (286), suggesting that infection-mediated disturbance in the host’s gut microbial community results in GIT malfunction.”