Differential Roles of Macrophages in Diverse Phases of Skin Repair

By T. Lucas, A. Waisman, R. Ranjan, J. Roes, T. Krieg, Werner Müller, Axel Roers, S. Eming. Journal of Immunology, 2010

1,157 citations recorded by the source corpus.

View Paper: https://www.semanticscholar.org/paper/61b990fe63d2da6b112c52a1097ac8b9fbcd4a75

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Citations in the corpus

Wound repair and regeneration: Mechanisms, signaling, and translation (2014, neutral): “…is that early-recruited neutrophils largely deal with killing invasive microorganisms at any breach of the skin barrier (102), whereas macrophages are needed for clearing apoptotic neutrophils and orchestrating early wound closure events, but also emit signals that cause later scarring (102, 103).”
INTERLEUKIN-4- AND INTERLEUKIN-13-MEDIATED ALTERNATIVELY ACTIVATED MACROPHAGES: ROLES IN HOMEOSTASIS AND DISEASE (2013, neutral): “For example, recent work confirming earlier observations (169, 170) indicates that normal wound healing responses are critically dependent on macrophages present during the first several days following sterile tissue damage such as surgical trauma.”
Modulation of macrophage phenotype by cell shape (2013, background): “Although distinct macrophage subpopulations are clearly observed at different phases of the immune response to infection and injury (2), regulation of phenotypic polarization of these remarkably plastic cells still remains poorly defined.”
Inflammation and wound healing: The role of the macrophage (2011, neutral): “15).”
Macrophage activation and its role in repair and pathology after spinal cord injury. (2015, background): “Depletion studies similar to the ones examining the role of macrophages during different phases of repair after skin injury have also not been performed after SCI (Lucas et al., 2010).”
Macrophage phenotypes during tissue repair (2013, background): “Mp, in particular, are known to be essential to regeneration, repair, and remodeling in numerous tissues [1–5].”
Macrophage plasticity and polarization: in vivo veritas. (2012, neutral): “During the early stages of the repair response after wounding the skin, infiltrating macrophages have an M2 phenotype and their depletion inhibits the formation of a highly vascularized, cellular granulation tissue and of scar tissues (36).”