Macrophage phenotypes during tissue repair
By Margaret L. Novak, T. Koh. Journal of Leukocyte Biology, 2013
623 citations recorded by the source corpus.
Subfields: Immunology, Molecular Biology, Rehabilitation
View Paper: https://www.semanticscholar.org/paper/32d24c1f92ce6e25fe1a62057f0c92ea3227742b
How researchers see this paper
The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.
In Immunology
- Paulina Krzyszczyk
- Andre F. Palmer
- G. Harry
- Francois Berthiaume
- R. Schloss
- C. McPherson
- Rubén Orihuela
- Tamás Rőszer
In Molecular Biology
In Rehabilitation
Citations in the corpus
- Macrophage Phenotypes Regulate Scar Formation and Chronic Wound Healing (2017, background): “M2 cells have been divided into four discrete types—M2a, M2b, M2c and M2d—in vitro based on their function and key markers (Table 1) [12].”
- Titanium Surface Characteristics, Including Topography and Wettability, Alter Macrophage Activation (2015, background): “Macrophages are responsible for the initial immune response, inflammation, and maintaining tissue homeostasis [3, 8, 9].”
- Understanding the Mysterious M2 Macrophage through Activation Markers and Effector Mechanisms (2015, background): “In response to the stimulus they sense, macrophages are being activated, which allows them to combat the pathogens, exert an immunomodulatory role, and maintain tissue integrity [4, 8, 9].”
- The Role of Macrophages in Acute and Chronic Wound Healing and Interventions to Promote Pro-wound Healing Phenotypes (2018, background): “In in vivo studies, this nomenclature is not as widely used to identify macrophages, potentially due to the heterogeneous populations present, which are generated from a variety of stimuli within wounds (Novak and Koh, 2013).”
- Microglial M1/M2 polarization and metabolic states (2016, background): “While the initial response of macrophages to injury has been known for some time, positive influences on tissue remodelling have been recognized more recently (Longbrake et al., 2007; Ruffell et al., 2009; Deng et al., 2012; Novak and Koh, 2013; Shechter and Schwartz, 2013; Shechter et al., 2013).”
- The Biology of Regeneration Failure and Success After Spinal Cord Injury. (2018, background): “Activated immune cells additionally contribute to neuronal and glial cell death by generating ROS through increased NADPH oxidase activity (132, 288).”