P3–370: APOE genotype has no influence in platelet and erythrocyte oxidative state

By M. Smith, C. Rottkamp, A. Nunomura, A. Raina, George Perry. 2006

834 citations recorded by the source corpus.

Subfields: Cardiology and Cardiovascular Medicine, Neurology, Physiology

View Paper: https://www.semanticscholar.org/paper/5d5e6240e237046d5d3d8979c93cf5aef3e004a4

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Neurology

1. M. Duennwald
2. Vy Ngo
3. E. Niki
4. Karen M. Rodrigue
5. Naftali Raz

In Physiology

1. Xiongwei Zhu
2. George Bartzokis
3. K. Ono
4. M. Duennwald
5. Vy Ngo
6. D. Selkoe
7. E. Niki
8. P. Reddy

Citations in the corpus

• Curcumin has potent anti‐amyloidogenic effects for Alzheimer's β‐amyloid fibrils in vitro (2004, background): “Many studies have demonstrated that oxidative damage is closely associated with the hallmark pathologies of AD and may play a critical role in the development of AD (Pratico and Delanty, 2000; Rottkamp et al., 2000; Smith et al., 2000; Varadarajan et al., 2000).”
• Neuropathology and Pathogenesis of Encephalitis following Amyloid β Immunization in Alzheimer's Disease (2004, background): “βA4 amyloid in senile plaques causes oxidative stress (3, 27, 34, 44), and oxidative stress is accompanied by regulatory responses of anti-oxidant molecules such as SOD-1 expression around senile plaques (17, 32).”
• Age-related myelin breakdown: a developmental model of cognitive decline and Alzheimer's disease. (2004, background): “[218] Smith MA, Rottkamp CA, Nunomura A, Raina AK, Perry G.”
• Nrf2 and Oxidative Stress: A General Overview of Mechanisms and Implications in Human Disease (2022, background): “Oxidative damage has been implicated in all major neurodegenerative diseases including Alzheimer’s disease (AD) [108,109], Parkinson’s disease (PD) [110,111], Huntington’s disease (HD) [112,113], amyotrophic lateral sclerosis (ALS) [114], and multiple sclerosis (MS) [115].”
• Involvement of Oxidative Stress in Alzheimer Disease (2006, background): “Oxidative modifications are further seen in cerebral tissue in early stages of AD, whereas oxidation of nucleic acids, proteins, and lipids are all prominent and early changes in AD, and oxidatively modified macromolecules are present in biologic fluids from the patients with AD as well as cellular ”