Update on acquired tetracycline resistance genes.
By M. Roberts. FEMS Microbiology Letters, 2005
992 citations recorded by the source corpus.
Subfields: Molecular Medicine, Pharmacology, Pollution
View Paper: https://www.semanticscholar.org/paper/5c58f0b18e4832804419b27ad71998083f03ed6e
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Citations in the corpus
- Efflux-Mediated Drug Resistance in Bacteria: an Update (2009, background): “Several Lactobacillus species derived from broiler chickens displayed tetracycline resistance due to the presence of the efflux genes tet(K), tet(L) or tet(Z), and the ribosomal protection genes tet(M) or tet(W).([512,513]) A tetracycline-resistant L.”
- Antibiotic Resistance Mechanisms in Bacteria: Relationships Between Resistance Determinants of Antibiotic Producers, Environmental Bacteria, and Clinical Pathogens (2018, background): “The acquired antibiotic efflux determinants, often found on MGEs in clinical isolates, are exemplified by many different types of tet genes (at least 22 have been identified) located on plasmids in both Gram-negative and Gram-positive bacteria (Roberts, 2005).”
- Acquired Antibiotic Resistance Genes: An Overview (2011, background): “There are currently over 40 different acquired tetracycline resistance determinants recognized, i.e., 38 tet (tetracycline resistance) and 3 otr (oxytetracycline resistance) genes, additionally 1 tcr gene has been identified (Roberts, 1996, 2005; Brown et al., 2008; see Table 6).”
- Antimicrobial Resistance in Bacteria: Mechanisms, Evolution, and Persistence (2019, neutral): “2003; Roberts 2005).”
- Efflux-mediated antimicrobial resistance. (2005, positive): “The vastmajorityof the different Tet proteins have been reported in Gram-negative bacteria (includes the Tet proteins A–E, G, H, I, J, Z and Tet(30) which are found exclusively in Gram-negative bacteria(47,89)) with two, TetK and TetL, found predominantly in Gram-positive bacteria.(89) Novel Tet eff”
- Mechanisms of Antibiotic Resistance (2016, background): “Many of these pumps affect tetracycline and doxycycline but do not decrease minocycline or tigecycline susceptibility, because they are not able to use these compounds as substrates (44, 47).”