GABA: a pioneer transmitter that excites immature neurons and generates primitive oscillations.

By Yehezkel Ben-Ari, J. Gaiarsa, R. Tyzio, Rustem Khazipov. Physiological Reviews, 2007

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Ion Channels in Innate and Adaptive Immunity (2015, result): “This is similar to the inhibitory mechanism by GABAA receptors in immature neurons (238, 239).”
Cation-chloride cotransporters and neuronal function. (2009, background): “…depolarizing extrasynaptic and synaptic GABAergic signaling promotes action potential activity, opening of voltage-gated Ca 2+ channels, and activation of NMDA receptors (Ben-Ari et al., 2007; Fukuda et al., 1998; Hennou et al., 2002; Kandler and Gillespie, 2005; Serafini et al., 1995).”
Brain development in rodents and humans: Identifying benchmarks of maturation and vulnerability to injury across species (2013, background): “KCC2 expression increases towards the end of the second postnatal week in the rat cortex (Ben-Ari et al., 2007; Tyzio et al., 2007), and at about 40 weeks post-conception in the human cortex (Dzhalaetal., 2005), in parallel with a decrease in spontaneous transients indicative of the ongoing…”
How Inhibition Shapes Cortical Activity (2011, positive): “In certain instances, for example in immature neurons (Ben-Ari et al., 2007) or in specialized neuronal compartments (Gulledge and Stuart, 2003; Szabadics et al., 2006; Woodruff et al., 2009), the reversal potential for Cl À is so depolarized that it may lead to an excitatory action of GABA A…”