Chemokines as regulators of T cell differentiation
By S. Luther, J. Cyster. Nature Immunology, 2001
777 citations recorded by the source corpus.
Subfields: Immunology, Oncology
View Paper: https://www.semanticscholar.org/paper/579fee0e92ccc0e9f98929303365e9d94761784d
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In Immunology
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In Oncology
Citations in the corpus
- Three or more routes for leukocyte migration into the central nervous system (2003, background): “We are doubly challenged, first, by the complexity of the vascular anatomy of the CNS and, second, by the many functions of molecules that are under study, many of which have properties well beyond those involved in traffickin”
- Lymphocyte traffic control by chemokines (2001, background): “Lymphocyte traffic control is the main task of chemokines but there is increasing evidence for migration-unrelated responses that are also modulated by chemokines, including lymphocyte differentiation and effector functions (see the review by Cyster in this issue of Nature Immunology”
- Unphosphorylated STAT3 accumulates in response to IL-6 and activates transcription by binding to NFkappaB. (2007, background): “For example, Ng et al. (2006) have shown that STAT3 is phosphorylated on Ser727 but not Tyr705 in response to activation of the TrkA receptor by nerve growth factor, and that serinephosphorylated STAT3 is important in driving signaldependent gene expression.”
- Structure, function, and inhibition of chemokines. (2002, background): “Nuclear magnetic resonance solution structure of truncated human GRObeta [5–73] and its structural comparison with CXC chemokine family members GROalpha and IL-8.”
- Cold Tumors: A Therapeutic Challenge for Immunotherapy (2019, background): “Chemokines acting on iDCs are the Monocyte Chemoattractant Proteins (CCL2, CCL7, CCL8) as well as CCL3/MIP-1alpha, CCL5/RANTES, and CCL4/MIP-1beta (44).”
- Monocyte emigration from bone marrow during bacterial infection requires signals mediated by chemokine receptor CCR2 (2006, background): “Monocyte chemoattractant protein 1 (MCP-1) is the main ligand for CCR2 (ref”