Lymphocyte traffic control by chemokines
By B. Moser, P. Loetscher. Nature Immunology, 2001
1,332 citations recorded by the source corpus.
Subfields: Immunology, Oncology
View Paper: https://www.semanticscholar.org/paper/5448e451248cf67aec2602c7b37dd2f4890366a9
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In Oncology
Citations in the corpus
- Chemokines: immunology's high impact factors (2001, neutral): “This subject is also discussed elsewhere in this issue of Nature Immunology”
- CXC chemokines in angiogenesis (2000, background): “Although these CXCR3 ligands have been shown to bind to CXCR3 on mononuclear cells [16,93,95–99], the ability of these non-ELR+ CXC chemokines to inhibit angiogenesis and induce lymphoma regression in immunoincompetent mice support the notion that these chemokines can mediate their effects in a T-ce”
- Inflammatory responses and inflammation-associated diseases in organs (2015, background): “Chemokines recruit inflammatory leukocytes to the infarct, and cytokines promote leukocyte-endothelial cell adhesions [104, 105].”
- Cytokines and Chemokines at the Crossroads of Neuroinflammation, Neurodegeneration, and Neuropathic Pain (2013, background): “Chemokines belong to two categories based on their biological activity, namely, the maintenance of homeostasis and the induction of inflammation [4].”
- Regulation of CXCR4 signaling. (2007, background): “These and other studies reveal that CXCR4 is essential for development, hematopoeisis, organogenesis, and vascularization [10–15], in addition to functioning as a classical chemokine receptor in the adult [16,17].”
- Vaccines: Effector and memory T-cell differentiation: implications for vaccine development (2002, positive): “Third, memory CD8 + T cells express a different pattern of surface proteins that are involved in cell adhesion and chemotaxis from naive T cells, which allows memory T cells to extravasate into non-lymphoid tissues and mucosal sites (for a review, see Ref”