The uncoupling protein homologues: UCP1, UCP2, UCP3, StUCP and AtUCP.

By D. Ricquier, F. Bouillaud. Biochemical Journal, 2000

837 citations recorded by the source corpus.

Subfields: Epidemiology, Physiology, Rehabilitation

View Paper: https://www.semanticscholar.org/paper/5792aa91653a05488476ffadf1c763d85f13dc4c

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Epidemiology

1. Gerald I. Shulman
2. D. Clegg
3. B. Palmer

In Physiology

1. Gerald I. Shulman
2. B. Kahn
3. Haiming Cao
4. V. Samuel
5. Ralph DeFronzo
6. K. Petersen
7. D. Clegg
8. B. Palmer

In Rehabilitation

1. Gerald I. Shulman

Citations in the corpus

• Sirt1 Regulates Insulin Secretion by Repressing UCP2 in Pancreatic β Cells (2005, neutral): “Several mammalian UCP homologues have been identified and characterized [28].”
• Disruption of the uncoupling protein-2 gene in mice reveals a role in immunity and reactive oxygen species production (2000, background): “Other reports have described a relationship between mitochondrial uncoupling and production of ROS (ref”
• The mitochondrial transporter family (SLC25): physiological and pathological implications (2004, neutral): “18, 19, 20, 21] or on particular MCs [22, 23, 24, 25, 26, 27].”
• Mitochondrial electron transport chain, ROS generation and uncoupling (Review) (2019, background): “UcP2 and UcP3, paralogues of UcP1, exhibit ~60% sequence identity with UcP1 and ~70% identity with each other (170,171).”
• Role of ROS and RNS Sources in Physiological and Pathological Conditions (2016, background): “However, whether in tissues differently from BAT UCP1 homologues are responsible for mitochondrial basal or inducible proton conductance is yet controversial [280].”
• Adaptations of Glucose and Long-Chain Fatty Acid Metabolism in Liver of Dairy Cows during the Periparturient Period (2001, background): “Recently, an inducible uncoupling protein, UCP-2, has been identified in tissues other than brown adipose tissue of rodents (Ricquier and Bouillaud, 2000).”
• Increased Energy Expenditure, Decreased Adiposity, and Tissue-Specific Insulin Sensitivity in Protein-Tyrosine Phosphatase 1B-Deficient Mice (2000, background): “Uncoupling can be promoted by increased expression of UCPs in brown fat and/or skeletal muscle, the major thermogenic tissues in rodents (14, 16, 31, 58).”