Chemokines: immunology's high impact factors

By C. Mackay. Nature Immunology, 2001

913 citations recorded by the source corpus.

Subfields: Immunology, Oncology

View Paper: https://www.semanticscholar.org/paper/2d535cef4a8fbc785ad58be56a299c7371a09cbf

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Immunology

1. J. Wong
2. I. Clark‐Lewis
3. A. Ben-Baruch
4. Simon A. Jones
5. Elias J. Fernandez
6. J. Wallace
7. Christopher M. Overall
8. G. A. McQuibban

In Oncology

1. Christopher M. Overall
2. J. Wong
3. I. Clark‐Lewis
4. A. Ben-Baruch
5. Simon A. Jones
6. Elias J. Fernandez
7. J. Wallace
8. G. A. McQuibban

Citations in the corpus

• Directing Transition from Innate to Acquired Immunity: Defining a Role for IL-61 (2005, background): “Significantly, IL-6 deficiency was associated with a disruption in the expression of chemokine receptors defined as Th1 (CXCR3, CCR5) and Th2 (CCR3, CCR4) subset markers (68).”
• Crucial importance of PKC-β(I) in LFA-1–mediated locomotion of activated T cells (2001, background): “Active T cell migration at this stage can be further facilitated and directed by chemotactic stimul”
• Matrix metalloproteinase processing of monocyte chemoattractant proteins generates CC chemokine receptor antagonists with anti-inflammatory properties in vivo. (2002, methodology): “Figure 2 shows that MCP-1 is cleaved at Ala4-Ile5, designated MCP-1(5-76); by MMP-1; to a lesser extent (20% of total by densitometric analysis) by MMP-3; and to a very minor extent (5% of total) by MMP-8.”
• The inflammatory chemokines CCL2 and CCL5 in breast cancer. (2008, background): “As such, the expression of these chemokines is usually inducible by the inflammatory insult, and is shut-off at the resolution of inflammation [9–11].”
• Structure, function, and inhibition of chemokines. (2002, background): “Nuclear magnetic resonance solution structure of truncated human GRObeta [5–73] and its structural comparison with CXC chemokine family members GROalpha and IL-8.”
• CXCR4–SDF-1 Signalling, Locomotion, Chemotaxis and Adhesion (2003, background): “A growing body of evidence has accumulated that a major role in this process is played by chemokines, small pro-inflammatory cytokines involved in a variety of immune reactions including infection, inflammation and tissue repair (Mackay 2001, Thelen 2001).”