An introduction to TRP channels.
By I. S. Ramsey, M. Delling, D. Clapham, Ramsey Delling Clapham. Annual Review of Physiology, 2006
1,611 citations recorded by the source corpus.
Subfields: Cellular and Molecular Neuroscience, Nutrition and Dietetics, Sensory Systems
View Paper: https://www.semanticscholar.org/paper/538d55a1fc436c4063137658346f093fb450464b
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In Cellular and Molecular Neuroscience
In Nutrition and Dietetics
In Sensory Systems
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Citations in the corpus
- The Epithelial Cell-derived Atopic Dermatitis Cytokine TSLP Activates Neurons to Induce Itch (2013, neutral): “What are the molecules mediating PAR2-evoked SOCE in keratinocytes? Both ORAI and TRPC channels have been implicated in SOCE (Cahalan, 2009; Ramsey et al., 2006).”
- Chemosensation in C. elegans. (2006, background): “There are seven families of TRP channels (Ramsey et al., 2006).”
- Calcium and cancer: targeting Ca2+ transport (2007, background): “Individual isoforms of these proteins may differ in their cellular and tissue distribution and regulation by other signalling pathway”
- Mechanisms of sensory transduction in the skin (2007, background): “Almost all of these belong to the transient receptor potential (TRP) family of cation channel”
- TRP channels and pain. (2013, background): “Whereas TRP and voltage-gated channels are thought to share similar overall structures (Ramsey et al. 2006), toxins that modify gating have apparently evolved to target different domains, likely reflecting the relative importance of these domains to the gating process.”
- PIP2 is a necessary cofactor for ion channel function: how and why? (2008, background): “PIP2 dependence is also evident in the TRP channels (67), channels that are activated by many exogenous and endogenous ligands and physical stimuli (65).”
- Lysosomal Physiology (2015, background): “Ren, unpublished data), suggesting that it is not mediated by the voltage-activated H+ channel HV1 (70, 71).”