An association between migraine and cutaneous allodynia

By R. Burstein, D. Yarnitsky, I. Goor-Aryeh, B. Ransil, Z. Bajwa. Annals of Neurology, 2000

1,012 citations recorded by the source corpus.

Subfields: Psychiatry and Mental health, Radiology, Nuclear Medicine and Imaging, Sensory Systems

View Paper: https://www.semanticscholar.org/paper/518301f50e38e6257b55e503e81603989db54faa

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Psychiatry and Mental health

1. Clifford J. Woolf
2. P. Goadsby
3. R. Burstein
4. Elizabeth M. C. Hillman

In Radiology, Nuclear Medicine and Imaging

1. David A. Boas
2. Andrew K. Dunn
3. R. Burstein
4. Theodore J. Huppert
5. Maria Angela Franceschini
6. Simon Robert Arridge
7. Sungho Tak
8. Jong Chul Ye

In Sensory Systems

1. Clifford J. Woolf
2. P. Goadsby
3. R. Burstein

Citations in the corpus

• Central sensitization: Implications for the diagnosis and treatment of pain (2010, background): “during migraine attacks in, and sometimes beyond the area of referred pain [36,37].”
• Intrinsic brain activity triggers trigeminal meningeal afferents in a migraine model (2002, background): “Although there are no studies measuring the caliber or flow in dural vessels during migraine headache, sensitization does develop as part of an attac”
• Defeating migraine pain with triptans: A race against the development of cutaneous allodynia (2004, methodology): “Quantitative sensory testing techniques used to measure skin pain thresholds and determine absence or presence of allodynia were described elsewhere.(4) Using the method of limits to determine thermal pain thresholds,(16) we attached a 30 30mm thermode (TSA-2001; Medoc, Ramat-Yishai, Israel) to the ”
• PATHOPHYSIOLOGY OF MIGRAINE (2006, background): “About two thirds of patients report pain from nonnoxious stimuli, allodynia (Burstein et al, 2000; Selby and Lance, 1960).”
• The development of cutaneous allodynia during a migraine attack clinical evidence for the sequential recruitment of spinal and supraspinal nociceptive neurons in migraine. (2000, methodology): “…further, measured on all four sites at 1, 2 and 4 h from the onset of we used quantitative sensory testing (QST) techniques to the headache (i.e. the pain). compare cutaneous pain thresholds of patients during migraine with their pain thresholds in the absence of migraine (Burstein et al., 2000).”
• Pathophysiology of Migraine: A Disorder of Sensory Processing. (2017, background): “Preclinical studies have helped describe the neural basis for allodynia in migraine (135, 136).”
• Can we conquer pain? (2002, background): “Such a shift in the excitability of the CNS contributes to the hypersensitivity to non-painful stimuli after surgery or during migraine attacks, as well as the diffuse muscle tenderness of myofascial syndromes, and sensory abnormalities of the gastro-intestinal tract in non-cardiac chest pain and ir”