Phagocyte partnership during the onset and resolution of inflammation

By Oliver Soehnlein, L. Lindbom. Nature reviews. Immunology, 2010

1,093 citations recorded by the source corpus.

View Paper: https://www.semanticscholar.org/paper/4e502bb7ed2d80934a623d1d43fc1fb0f8b4b5ae

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In Immunology

Citations in the corpus

Cellular and molecular mechanisms of skin wound healing (2024, neutral): “Lipoxin also promotes resolution by inhibiting the influx of neutrophils into the wound and by promoting monocyte migration 120–122 and their eventual emigration to the draining lymph nodes 94 .”
Rhythmic modulation of the hematopoietic niche through neutrophil clearance (2013, positive): “In inflammatory scenarios, neutrophils recruited to injured tissues are eventually cleared by macrophages, a process that contributes to resolving inflammation and restoring homeostasis (Serhan and Savill, 2005; Soehnlein and Lindbom, 2010).”
Diverse novel functions of neutrophils in immunity, inflammation, and beyond (2013, background): “Apoptosis of neutrophils triggers an active process promoting resolution of inflammation (Serhan, 2007; Serhan et al., 2008; Soehnlein and Lindbom, 2010).”
Satellite Cells and Skeletal Muscle Regeneration (2015, positive): “Anti-inflammatory molecules and proresolving mediators released by these cells will stop the further recruitment of granulocytes and favor nonphlogistic phagocytosis (e.g., that does not stimulate inflammation) of apoptotic neutrophils (281).”
Pathophysiology of Myocardial Infarction (2015, positive): “Moreover, phagocytotic uptake of apoptotic neutrophils activates an anti-inflammatory program in macrophages inducing synthesis and release of IL-10, TGF-β , and of prore-solving lipid mediators (such as lipoxins and resolvins) (337).”
Neutrophils orchestrate post-myocardial infarction healing by polarizing macrophages towards a reparative phenotype (2016, background): “Macrophages engulfing apoptotic neutrophils activate an anti-inflammatory response by inhibiting pro-inflammatory cytokines and inducing the production of interleukin (IL)-10, transforming growth factor (TGF)-b and pro-resolving lipid mediators.(10) Consequently, anti-inflammatory strategies reducin”
Skin Acute Wound Healing: A Comprehensive Review (2019, background): “This myelomonocytic interaction is a key component of wound repair [62, 66, 75].”
Neuroinflammation and M2 microglia: the good, the bad, and the inflamed (2014, background): “to promote killing of any invading organism and remove dead cells to ‘clean’ the damaged area [62].”