Axon degeneration mechanisms: commonality amid diversity
By M. Coleman. Nature Reviews Neuroscience, 2005
858 citations recorded by the source corpus.
Subfields: Cellular and Molecular Neuroscience, Developmental Neuroscience, Neurology
View Paper: https://www.semanticscholar.org/paper/4dc774ca4c58c476dc77a3d8329d4224a5ef7954
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In Cellular and Molecular Neuroscience
In Neurology
Citations in the corpus
- Decreased white matter integrity in late-myelinating fiber pathways in Alzheimer's disease supports retrogenesis (2008, background): “However, many of these late-myelinating pathways also connect to medial temporal lobe structures affected early in AD (Brun and Englund,1986), so it is possible that observedWM changesmay reflect wallerian degeneration secondary to neuronal loss as well (see Coleman, 2005).”
- The secret life of NAD+: an old metabolite controlling new metabolic signaling pathways. (2010, background): “Also, in the central nervous system the Wld mouse was protected from pathological states, such as Parkinson disease (212, 217).”
- A reversible form of axon damage in experimental autoimmune encephalomyelitis and multiple sclerosis (2011, background): “Reversibility, spatial restriction and speed distinguish FAD from other forms of axon loss in development and diseas”
- NAD+/NADH and NADP+/NADPH in Cellular Functions and Cell Death: Regulation and Biological Consequences (2008, neutral): “Axonal degeneration is one of the major pathological changes in many neurodegenerative diseases (60).”