Cytochrome C-mediated apoptosis.

By Xuejun Jiang, Xiaodong Wang. Annual Review of Biochemistry, 2003

1,432 citations recorded by the source corpus.

Subfields: Molecular Biology, Nutrition and Dietetics

View Paper: https://www.semanticscholar.org/paper/4917bbbd4b930218a5d10084e73bf462ef2b5787

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Citations in the corpus

Human inhibitor of apoptosis proteins: why XIAP is the black sheep of the family (2006, background): “The ‘intrinsic pathway’ senses intracellular damage and growth factor or cytokine availability, and activates caspase-9 at the apoptosome (Jiang & Wang, 2004).”
Cullin3-based polyubiquitination and p62-dependent aggregation of caspase-8 mediate extrinsic apoptosis signaling. (2009, neutral): “In the intrinsic pathway, the ‘‘apoptosome’’ mediates multimerization of the apical protease caspase-9, facilitating its full activation (Jiang and Wang, 2004; Riedl and Salvesen, 2007).”
Role of Mitochondria in Ferroptosis (2018, background): “In mitochondria-mediated apoptosis, the mitochondrion serves as a reservoir, storing apoptosis-regulatory proteins such as cytochrome c and Smac, which upon release to the cytoplasm, promote caspase activation (Jiang and Wang, 2004; Wang, 2001).”
Histone deacetylase inhibitors: molecular mechanisms of action (2007, background): “It is regulated, in part, by proand antiapoptotic proteins of bcl-2 family (Jiang and Wang, 2004).”
Sexually dimorphic neurons in the ventromedial hypothalamus govern mating in both sexes and aggression in males (2013, neutral): “Moreover, we establish that a discrete, sexually dimorphic neuronal population influences sexually dimorphic behaviors in both sexes.”
Mitochondrial Dysfunction in Parkinson's Disease (2011, background): “[11] X.”
MicroRNA: Biogenesis, Function and Role in Cancer (2010, background): “BCL2 functions to block apoptosis by inhibiting mitochondrial cytochrome C release necessary to activate the caspase pathway of enzymes responsible for directing programmed cell death [268, 269].”
Emerging mechanisms and disease relevance of ferroptosis (2020, background): “The consequence of this intercellular communication is in stark contrast to that of death receptor-mediated apoptosis [90,91], in which one cell (bearing the death receptor ligand) induces apoptotic death of the other (bearing the death receptor).”
Apoptotic caspases prevent the induction of type I interferons by mitochondrial DNA (2014, background): “the Induction of Type I Interferons by Mitochondrial DNA”